MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

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Modelling Cooperative Tumorigenesis in Drosophila.

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P2860

MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

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type

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MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

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MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

@en

P1476

MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes

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P2093

E Lyulcheva

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E Taylor

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L Dodgson

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N Alqadri

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P2860

Signal-regulated activation of serum response factor is mediated by changes in actin dynamics

Inflammation and cancer

Drosophila as a Potential Model for Ocular Tumors

Lamellipodin promotes invasive 3D cancer cell migration via regulated interactions with Ena/VASP and SCAR/WAVE.

A drosophila model for EGFR-Ras and PI3K-dependent human glioma

Actin filament turnover drives leading edge growth during myelin sheath formation in the central nervous system.

Coordinated regulation of actin filament turnover by a high-molecular-weight Srv2/CAP complex, cofilin, profilin, and Aip1.

Regulators of actin filament barbed ends at a glance

Systematic variation in gene expression patterns in human cancer cell lines

OMERO: flexible, model-driven data management for experimental biology

P2888

onc.2017.68

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4311-4322

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scholarly article

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10.1038/ONC.2017.68

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English

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Giovanni Messina

David Mason

Daimark Bennett

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2017-03-27T00:00:00Z

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1084129948

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28346426

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glia

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5537612

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MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

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P2860

P2860

MRL proteins cooperate with activated Ras in glia to drive distinct oncogenic outcomes.

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