Early enhanced local neutrophil recruitment in peritonitis-induced sepsis improves bacterial clearance and survival.
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Streptococcus pyogenes SpyCEP influences host-pathogen interactions during infection in a murine air pouch modelHyperresponsiveness of mice deficient in plasma-secreted sphingomyelinase reveals its pivotal role in early phase of host responseCelecoxib Enhances the Efficacy of Low-Dose Antibiotic Treatment against Polymicrobial Sepsis in Mice and Clinical Isolates of ESKAPE Pathogens.Nonhematopoietic β-Arrestin-1 inhibits inflammation in a murine model of polymicrobial sepsis.Depletion of dendritic cells enhances innate anti-bacterial host defense through modulation of phagocyte homeostasis.Modulation of chemokine gradients by apheresis redirects leukocyte trafficking to different compartments during sepsis, studies in a rat model.Role of surfactant proteins A and D in sepsis-induced acute kidney injury.Hypertonic saline solution drives neutrophil from bystander organ to infectious site in polymicrobial sepsis: a cecal ligation and puncture model.Anti-inflammatory effect of a retrovirus-derived immunosuppressive peptide in mouse models.Role of myeloid-specific G-protein coupled receptor kinase-2 in sepsis.NK cells require IL-28R for optimal in vivo activityPresence of preexisting antibodies mediates survival in sepsis.IL-15 Superagonist Expands mCD8+ T, NK and NKT Cells after Burn Injury but Fails to Improve Outcome during Burn Wound InfectionTLR4-Dependent Secretion by Hepatic Stellate Cells of the Neutrophil-Chemoattractant CXCL1 Mediates Liver Response to Gut Microbiota.Acute-Phase Deaths from Murine Polymicrobial Sepsis Are Characterized by Innate Immune Suppression Rather Than ExhaustionCecal ligation and puncture-induced murine sepsis does not cause lung injury.High Mobility Group Box-1 mediates hyperoxia-induced impairment of Pseudomonas aeruginosa clearance and inflammatory lung injury in mice.Fish Oil-Based Fat Emulsion Reduces Acute Kidney Injury and Inflammatory Response in Antibiotic-Treated Polymicrobial Septic Mice.Lipopolysaccharide clearance, bacterial clearance, and systemic inflammatory responses are regulated by cell type-specific functions of TLR4 during sepsis.Pathophysiology of septic shock: From bench to bedside.Chemokine CXCL1 mediated neutrophil recruitment: Role of glycosaminoglycan interactions.AQP5-1364A/C polymorphism and the AQP5 expression influence sepsis survival and immune cell migration: a prospective laboratory and patient study.Sepsis: multiple abnormalities, heterogeneous responses, and evolving understanding.Noradrenergic neurons regulate monocyte trafficking and mortality during gram-negative peritonitis in mice.The impact of environmental enrichment on the murine inflammatory immune response.Pathophysiologic mechanisms in septic shock.Effect of Curcumin in Experimental Peritonitis.The Role of Substance P in Pulmonary Clearance of Bacteria in Comparative Injury Models.Antagonism of the Neurokinin-1 Receptor Improves Survival in a Mouse Model of Sepsis by Decreasing Inflammation and Increasing Early Cardiovascular Function.Deletion of Nlrp3 Augments Survival during Polymicrobial Sepsis by Decreasing Autophagy and Enhancing Phagocytosis.Characterization of changes in plasma and tissue oxylipin levels in LPS and CLP induced murine sepsis.Human Neutrophils Support Thrombin Generation in Peritonitis.Alpha-Lipoic Acid Attenuates Oxidative Damage in Organs After Sepsis.Early murine polymicrobial sepsis predominantly causes renal injury.Pyroptosis of resident macrophages differentially orchestrates inflammatory responses to Staphylococcus aureus in resistant and susceptible mice.Diminished neutrophil extracellular trap (NET) formation is a novel innate immune deficiency induced by acute ethanol exposure in polymicrobial sepsis, which can be rescued by CXCL1G-protein-coupled receptor kinase-5 mediates inflammation but does not regulate cellular infiltration or bacterial load in a polymicrobial sepsis model in mice.CXCL1 contributes to host defense in polymicrobial sepsis via modulating T cell and neutrophil functions.Identification of B7-H1 as a novel mediator of the innate immune/proinflammatory response as well as a possible myeloid cell prognostic biomarker in sepsis.NF-κB inhibition after cecal ligation and puncture reduces sepsis-associated lung injury without altering bacterial host defense.
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P2860
Early enhanced local neutrophil recruitment in peritonitis-induced sepsis improves bacterial clearance and survival.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Early enhanced local neutrophi ...... terial clearance and survival.
@en
type
label
Early enhanced local neutrophi ...... terial clearance and survival.
@en
prefLabel
Early enhanced local neutrophi ...... terial clearance and survival.
@en
P2860
P356
P1476
Early enhanced local neutrophi ...... cterial clearance and survival
@en
P2093
Elizabeth R Schuller
Florin L Craciun
P2860
P304
P356
10.4049/JIMMUNOL.1002300
P407
P577
2010-11-01T00:00:00Z