Long-term productive human cytomegalovirus infection of a human neuroblastoma cell line.
about
Oncomodulation by human cytomegalovirus: novel clinical findings open new roads.Human cytomegalovirus infection in tumor cells of the nervous system is not detectable with standardized pathologico-virological diagnostics.The story of human cytomegalovirus and cancer: increasing evidence and open questions.Chemoresistance acquisition induces a global shift of expression of aniogenesis-associated genes and increased pro-angogenic activity in neuroblastoma cells.Human cytomegalovirus induces apoptosis in neural stem/progenitor cells derived from induced pluripotent stem cells by generating mitochondrial dysfunction and endoplasmic reticulum stress.miR-137 regulates the constitutive androstane receptor and modulates doxorubicin sensitivity in parental and doxorubicin-resistant neuroblastoma cellsMaintenance of large numbers of virus genomes in human cytomegalovirus-infected T98G glioblastoma cellsHuman cytomegalovirus infection of tumor cells downregulates NCAM (CD56): a novel mechanism for virus-induced tumor invasiveness.Cytotoxic potential of IL-15-activated cytokine-induced killer cells against human neuroblastoma cells.Frequent detection of human cytomegalovirus in neuroblastoma: a novel therapeutic target?Selection of a highly invasive neuroblastoma cell population through long-term human cytomegalovirus infection.Mechanisms of ellipticine-mediated resistance in UKF-NB-4 neuroblastoma cells.Functional analysis of the human cytomegalovirus US28 gene by insertion mutagenesis with the green fluorescent protein gene.Tumor-endothelium cross talk blocks recruitment of neutrophils to endothelial cells: a novel mechanism of endothelial cell anergy.Human cytomegalovirus productively infects adrenocortical cells and induces an early cortisol response.Long-term infection and shedding of human cytomegalovirus in T98G glioblastoma cells.Human cytomegalovirus infection alters PC3 prostate carcinoma cell adhesion to endothelial cells and extracellular matrix.Human cytomegalovirus (HCMV) IE1 plays role in resistance to apoptosis with etoposide in cancer cell line by Cdk2 accumulation.Expression level of neural cell adhesion molecule (NCAM) inversely correlates with the ability of neuroblastoma cells to adhere to endothelium in vitro.Human cytomegalovirus infection decreases expression of thrombospondin-1 independent of the tumor suppressor protein p53.Testing of SNS-032 in a Panel of Human Neuroblastoma Cell Lines with Acquired Resistance to a Broad Range of Drugs.Towards an unbiased, collaborative effort to reach evidence about the presence of human cytomegalovirus in glioblastoma (and other tumors).The Human Cytomegalovirus, from Oncomodulation to Oncogenesis
P2860
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P2860
Long-term productive human cytomegalovirus infection of a human neuroblastoma cell line.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Long-term productive human cytomegalovirus infection of a human neuroblastoma cell line.
@en
type
label
Long-term productive human cytomegalovirus infection of a human neuroblastoma cell line.
@en
prefLabel
Long-term productive human cytomegalovirus infection of a human neuroblastoma cell line.
@en
P2093
P2860
P1476
Long-term productive human cytomegalovirus infection of a human neuroblastoma cell line.
@en
P2093
B Kornhuber
P2860
P356
10.1002/(SICI)1097-0215(19960103)65:1<90::AID-IJC16>3.0.CO;2-M
P577
1996-01-01T00:00:00Z