YM155 as an inhibitor of cancer stemness simultaneously inhibits autophosphorylation of epidermal growth factor receptor and G9a-mediated stemness in lung cancer cells.

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STAT3 exacerbates survival of cancer stem-like tumorspheres in EGFR-positive colorectal cancers: RNAseq analysis and therapeutic screening

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Q58803100-5D701BEB-05C6-4816-9761-A535DFF206FD

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YM155 as an inhibitor of cancer stemness simultaneously inhibits autophosphorylation of epidermal growth factor receptor and G9a-mediated stemness in lung cancer cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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YM155 as an inhibitor of cance ...... stemness in lung cancer cells.

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YM155 as an inhibitor of cance ...... stemness in lung cancer cells.

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YM155 as an inhibitor of cance ...... stemness in lung cancer cells.

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P1476

YM155 as an inhibitor of cance ...... stemness in lung cancer cells.

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P2093

Ai-Sheng Ho

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Cheng-Wen Wu

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Chun-Chao Chang

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Huan-Chau Lin

http://www.w3.org/2001/XMLSchema#string

Jungshan Chang

http://www.w3.org/2001/XMLSchema#string

Ken-Hong Lim

http://www.w3.org/2001/XMLSchema#string

Ling Huang

http://www.w3.org/2001/XMLSchema#string

Stanley Ching-Cheng Huang

http://www.w3.org/2001/XMLSchema#string

Yi-Fang Chang

http://www.w3.org/2001/XMLSchema#string

Yu-Cheng Chang

http://www.w3.org/2001/XMLSchema#string

P2860

Formation of pluripotent stem cells in the mammalian embryo depends on the POU transcription factor Oct4

G9a and Glp methylate lysine 373 in the tumor suppressor p53

G9a histone methyltransferase plays a dominant role in euchromatic histone H3 lysine 9 methylation and is essential for early embryogenesis

EGFR mutation and resistance of non-small-cell lung cancer to gefitinib.

MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling.

Phosphorylated EGFR expression may predict outcome of EGFR-TKIs therapy for the advanced NSCLC patients with wild-type EGFR

Activating mutations in the epidermal growth factor receptor underlying responsiveness of non-small-cell lung cancer to gefitinib

Set domain-containing protein, G9a, is a novel lysine-preferring mammalian histone methyltransferase with hyperactivity and specific selectivity to lysines 9 and 27 of histone H3

Interleukin enhancer-binding factor 3/NF110 is a target of YM155, a suppressant of survivin

Histone methyltransferases G9a and GLP form heteromeric complexes and are both crucial for methylation of euchromatin at H3-K9

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e0182149

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scholarly article

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10.1371/JOURNAL.PONE.0182149

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English

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Chun-Chia Cheng

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2017-08-07T00:00:00Z

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28787001

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5546577

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YM155 as an inhibitor of cancer stemness simultaneously inhibits autophosphorylation of epidermal growth factor receptor and G9a-mediated stemness in lung cancer cells.

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P2860

P2860

YM155 as an inhibitor of cancer stemness simultaneously inhibits autophosphorylation of epidermal growth factor receptor and G9a-mediated stemness in lung cancer cells.

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