Knockdown of Tlr4 in the Arcuate Nucleus Improves Obesity Related Metabolic Disorders

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Knockdown of Tlr4 in the Arcuate Nucleus Improves Obesity Related Metabolic Disorders

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2017 nî lūn-bûn

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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Knockdown of Tlr4 in the Arcuate Nucleus Improves Obesity Related Metabolic Disorders

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Knockdown of Tlr4 in the Arcuate Nucleus Improves Obesity Related Metabolic Disorders

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Knockdown of Tlr4 in the Arcuate Nucleus Improves Obesity Related Metabolic Disorders

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P1476

Knockdown of Tlr4 in the Arcuate Nucleus Improves Obesity Related Metabolic Disorders

@en

P2093

Guohua Li

http://www.w3.org/2001/XMLSchema#string

Ying Li

http://www.w3.org/2001/XMLSchema#string

Yongli Zhao

http://www.w3.org/2001/XMLSchema#string

Yuchuan Wang

http://www.w3.org/2001/XMLSchema#string

Zhengjuan Liu

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P2860

Obesity and insulin resistance

Neural control of energy balance: translating circuits to therapies

Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity

Lentiviral vector-mediated knockdown of Lrb in the arcuate nucleus promotes diet-induced obesity in rats.

Toll-like receptor 4 in CNS pathologies

An inhibitor of the protein kinases TBK1 and IKK-ɛ improves obesity-related metabolic dysfunctions in mice

Brain innate immunity regulates hypothalamic arcuate neuronal activity and feeding behavior.

TLR4 at the Crossroads of Nutrients, Gut Microbiota, and Metabolic Inflammation.

Remodeling of the arcuate nucleus energy-balance circuit is inhibited in obese mice.

Increased hypothalamic inflammation associated with the susceptibility to obesity in rats exposed to high-fat diet

P2888

s41598-017-07858-6

P304

7441

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P31

scholarly article

P356

10.1038/S41598-017-07858-6

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-08-07T00:00:00Z

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P698

28785099

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P921

Adipositas

Toll-like receptor

P932

5547063

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