Bcl-x and Bcl-2 inhibit TNF and Fas-induced apoptosis and activation of phospholipase A2 in breast carcinoma cells.
about
Gene therapy for carcinoma of the breast: Pro-apoptotic gene therapyExcess sphingomyelin disturbs ATG9A trafficking and autophagosome closure.Potential dengue virus-triggered apoptotic pathway in human neuroblastoma cells: arachidonic acid, superoxide anion, and NF-kappaB are sequentially involved.Identification of cytoskeleton-associated proteins essential for lysosomal stability and survival of human cancer cellsA novel gene coding for a Fas apoptosis inhibitory molecule (FAIM) isolated from inducibly Fas-resistant B lymphocytesTwo CD95 (APO-1/Fas) signaling pathwaysCathepsin B acts as a dominant execution protease in tumor cell apoptosis induced by tumor necrosis factor.Apoptin-induced cell death is modulated by Bcl-2 family members and is Apaf-1 dependent.Human mammary fibroblasts stimulate invasion of breast cancer cells in a three-dimensional culture and increase stroma development in mouse xenograftsHsp70 exerts its anti-apoptotic function downstream of caspase-3-like proteases.CD40 induces apoptosis in carcinoma cells through activation of cytotoxic ligands of the tumor necrosis factor superfamilyBid, a widely expressed proapoptotic protein of the Bcl-2 family, displays lipid transfer activity.Oxidative stress and autophagy in the regulation of lysosome-dependent neuron death.Contrasting effects of matrix protein on apoptosis in HeLa and BHK cells infected with vesicular stomatitis virus are due to inhibition of host gene expression.Apoptosome-independent activation of the lysosomal cell death pathway by caspase-9.Selective depletion of heat shock protein 70 (Hsp70) activates a tumor-specific death program that is independent of caspases and bypasses Bcl-2.Enhanced lymphocyte longevity and absence of proliferation and lymphocyte apoptosis in Quilty effects of human heart allografts.The adenovirus E3-14.7K protein and the E3-10.4K/14.5K complex of proteins, which independently inhibit tumor necrosis factor (TNF)-induced apoptosis, also independently inhibit TNF-induced release of arachidonic acid.Adenovirus E3-10.4K/14.5K protein complex inhibits tumor necrosis factor-induced translocation of cytosolic phospholipase A2 to membranes.The ability of BHRF1 to inhibit apoptosis is dependent on stimulus and cell type.CrmA expression in T lymphocytes of transgenic mice inhibits CD95 (Fas/APO-1)-transduced apoptosis, but does not cause lymphadenopathy or autoimmune disease.Major Alterations of Phosphatidylcholine and Lysophosphotidylcholine Lipids in the Substantia Nigra Using an Early Stage Model of Parkinson's Disease.Sensitive detection of lysosomal membrane permeabilization by lysosomal galectin puncta assay.Tetrahydrofurandiol stimulation of phospholipase A2, lipoxygenase, and cyclooxygenase gene expression and MCF-7 human breast cancer cell proliferationThe adenovirus E3/10.4K-14.5K proteins down-modulate the apoptosis receptor Fas/Apo-1 by inducing its internalization.The Fas counterattack: Fas-mediated T cell killing by colon cancer cells expressing Fas ligandCytokine response modifier A (CrmA) inhibits ceramide formation in response to tumor necrosis factor (TNF)-alpha: CrmA and Bcl-2 target distinct components in the apoptotic pathwayDendritic cell apoptosis and the pathogenesis of dengueThe Fas counterattack: a molecular mechanism of tumor immune privilege.The TRAIL to targeted therapy of breast cancer.Characterization of reaper- and FADD-induced apoptosis in a lepidopteran cell lineFractionated gamma-irradiation renders tumour cells more responsive to apoptotic signals through CD95.Regulation of ERalpha-mediated transcription of Bcl-2 by PI3K-AKT crosstalk: implications for breast cancer cell survival.Involvement of caspase-dependent activation of cytosolic phospholipase A2 in tumor necrosis factor-induced apoptosisAn apoptosis-inducing isoform of neu differentiation factor (NDF) identified using a novel screen for dominant, apoptosis-inducing genesProduct inhibition of secreted phospholipase A2 may explain lysophosphatidylcholines' unexpected therapeutic propertiesLack of cell surface Fas/APO-1 expression in pulmonary adenocarcinomas.The molecular biology of apoptosis.Protection against CD95-induced apoptosis by chlamydial infection at a mitochondrial step.Exogenous C2-ceramide activates c-fos serum response element via Rac-dependent signalling pathway.
P2860
Q24797919-AC6A0F93-FBFF-400F-A518-1BEE66B05AB5Q27311248-0EECD8A1-5E3C-471B-9015-49EAE7954695Q27470005-EEE1AA75-226F-48FC-8FA7-BF75A2732F2BQ28484373-61FC84DB-0C77-4B82-BCA7-B5D38647044AQ28587136-9587DAD5-FE70-469F-B141-7427B597B282Q29619631-2B261AE0-2849-42AD-B9A8-4C20D9E6503EQ30481000-69120CFC-B49F-4FE9-8E0B-6A656E0C0936Q33227197-C4FCBA02-D0E7-4C4C-879B-1D29945936C4Q33665522-0770C243-0322-47CE-8698-08523FE06F9EQ33889765-307D420E-711F-411D-B048-DBBFA1C5AD0BQ33964729-510CFABC-C20C-4F24-874F-3D41ECC0C14BQ34012615-BB168462-189B-4B23-B6F6-ECD119C9A027Q34104922-A1E9D3C4-B211-4285-99BC-42850D7F10EEQ34857976-C8754856-7661-467B-AF05-0491B3606D9CQ35131409-B2297D71-0951-480E-A8F5-0B333E271A2DQ35170422-7A9F1364-03D3-4997-BD69-73A70E87697DQ35763713-9E70E9BF-8C19-460F-B21C-F68DF207461CQ35865485-3C8BCB71-9BE7-4FF8-AF53-28CDD799CC67Q35880562-36717F84-C774-4EB3-A566-F95BD069D398Q35895541-E5590289-A32C-4BDB-8223-11CD97B962A2Q35909505-A9C13D2A-43D5-42F7-8C4D-F9E2BB1349C5Q36088639-3921BBC3-5E1B-415F-ADD7-42B4E4412F4EQ36111729-3409AA58-D8A1-474D-85C5-81CB7CBA2ED1Q36264601-52B67B80-C780-40D8-A7CE-8EEB1DE7A1ACQ36284758-1290E1E5-E944-4D96-A99A-7CE0A0DB4491Q36367323-CDDEA72F-C5EB-45A3-9A53-0E665387C52CQ36376514-E58B0D91-217A-4555-B301-ABC3F2C38F01Q36432712-CA681512-20E8-4C78-BF16-68BAA1262CCCQ36437768-0FCD12FF-2BBF-4732-B119-D44A0D5BA706Q36511919-1B765247-AFE2-411D-95AD-0A190F306639Q36565182-FE6774B7-22AE-4BD3-A7B5-B82BC6B644B0Q36620584-96870C7F-F984-4875-AD88-7CA530C5D82DQ36731304-7DE2C151-DC26-4444-9618-41EFFFBECF5CQ36765200-07A25400-6FEC-45AB-B5AC-8996CDB4A361Q36852573-CAB71155-991F-4D6F-BE81-03F3AF681DD7Q36967194-0856041C-6536-4137-9AD9-94C2F2C7E7AFQ37378833-45E93129-874E-4482-A331-EEA152BFD498Q37688810-FCF84427-FAF0-45A2-8CBC-1B5D356FD66CQ37867829-8A3DC5E8-8C91-4F6B-9134-591289432FBCQ38338513-811D06E2-4618-43BE-B061-B00350A29BAC
P2860
Bcl-x and Bcl-2 inhibit TNF and Fas-induced apoptosis and activation of phospholipase A2 in breast carcinoma cells.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Bcl-x and Bcl-2 inhibit TNF an ...... A2 in breast carcinoma cells.
@en
type
label
Bcl-x and Bcl-2 inhibit TNF an ...... A2 in breast carcinoma cells.
@en
prefLabel
Bcl-x and Bcl-2 inhibit TNF an ...... A2 in breast carcinoma cells.
@en
P2093
P1433
P1476
Bcl-x and Bcl-2 inhibit TNF an ...... e A2 in breast carcinoma cells
@en
P2093
P304
P407
P577
1995-06-01T00:00:00Z