A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.
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Hyperinsulinemia is Associated with Increased Soluble Insulin Receptors Release from HepatocytesReceptor cleavage reduces the fluid shear response in neutrophils of the spontaneously hypertensive ratMast Cell Proteases and InflammationProteolytic Activity Attenuates the Response of Endothelial Cells to Fluid Shear StressProteolytic receptor cleavage in the pathogenesis of blood rheology and co-morbidities in metabolic syndrome. Early forms of autodigestion.An emerging role of degrading proteinases in hypertension and the metabolic syndrome: autodigestion and receptor cleavage.The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.Proteolytic Cleavage of the Red Blood Cell Glycocalyx in a Genetic Form of Hypertension.Pancreatic digestive enzyme blockade in the small intestine prevents insulin resistance in hemorrhagic shock.Mechanisms of I/R-Induced Endothelium-Dependent Vasodilator Dysfunction.
P2860
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P2860
A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.
@en
type
label
A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.
@en
prefLabel
A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.
@en
P2093
P2860
P356
P1476
A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.
@en
P2093
Cuihua Zhang
Edward E Tran
Frank A Delano
Geert W Schmid-Schönbein
Hanrui Zhang
P2860
P304
P356
10.1586/EEM.09.64
P577
2010-01-01T00:00:00Z