Mutation of residues in the C3dg region of human complement component C3 corresponding to a proposed binding site for complement receptor type 2 (CR2, CD21) does not abolish binding of iC3b or C3dg to CR2.
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Structural insight on the recognition of surface-bound opsonins by the integrin I domain of complement receptor 3C3d adjuvant effects are mediated through the activation of C3d-specific autoreactive T cellsDissociation of HIV-1 from follicular dendritic cells during HAART: mathematical analysis.Contribution of follicular dendritic cells to persistent HIV viremia.Deglycosylation and label-free quantitative LC-MALDI MS applied to efficient serum biomarker discovery of lung cancer.
P2860
Mutation of residues in the C3dg region of human complement component C3 corresponding to a proposed binding site for complement receptor type 2 (CR2, CD21) does not abolish binding of iC3b or C3dg to CR2.
description
1995 nî lūn-bûn
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1995年の論文
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1995年論文
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1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
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1995年論文
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1995年论文
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1995年论文
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1995年论文
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name
Mutation of residues in the C3 ...... inding of iC3b or C3dg to CR2.
@en
type
label
Mutation of residues in the C3 ...... inding of iC3b or C3dg to CR2.
@en
prefLabel
Mutation of residues in the C3 ...... inding of iC3b or C3dg to CR2.
@en
P1476
Mutation of residues in the C3 ...... binding of iC3b or C3dg to CR2
@en
P2093
Isenman DE
P304
P407
P577
1995-03-01T00:00:00Z