Cardioprotective function of mitochondrial-targeted and transcriptionally inactive STAT3 against ischemia and reperfusion injury. - Wikidata - wikimedia - TriplyDB

Cardioprotective function of mitochondrial-targeted and transcriptionally inactive STAT3 against ischemia and reperfusion injury.

Cardioprotective function of mitochondrial-targeted and transcriptionally inactive STAT3 against ischemia and reperfusion injury.

http://www.wikidata.org/entity/Q41377233

about

Mitochondrial Stat3, the Need for Design Thinking Stress-induced dynamic regulation of mitochondrial STAT3 and its association with cyclophilin D reduce mitochondrial ROS production Sevoflurane postconditioning improves myocardial mitochondrial respiratory function and reduces myocardial ischemia-reperfusion injury by up-regulating HIF-1.Ischemic postconditioning influences electron transport chain protein turnover in Langendorff-perfused rat hearts.Tackling myocardial ischemic injury: the signal transducer and activator of transcription 3 (STAT3) at a good site.TNFR2 Stimulation Promotes Mitochondrial Fusion via Stat3- and NF-kB-Dependent Activation of OPA1 Expression.Mitochondrial STAT3 is negatively regulated by SOCS3 and upregulated after spinal cord injury.Manganese- and 1-methyl-4-phenylpyridinium-induced neurotoxicity display differences in morphological, electrophysiological and genome-wide alterations: implications for idiopathic Parkinson's disease.The SAFE pathway for cardioprotection: is this a promising target?

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Cardioprotective function of mitochondrial-targeted and transcriptionally inactive STAT3 against ischemia and reperfusion injury.

http://www.wikidata.org/entity/Q41377233

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Cardioprotective function of m ...... chemia and reperfusion injury.

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type

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Cardioprotective function of m ...... chemia and reperfusion injury.

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Cardioprotective function of m ...... chemia and reperfusion injury.

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S00395-015-0509-2

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Basic Research in Cardiology

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Cardioprotective function of m ...... chemia and reperfusion injury.

@en

P2093

Aijun Xu

http://www.w3.org/2001/XMLSchema#string

Andrew C Larner

http://www.w3.org/2001/XMLSchema#string

Edward J Lesnefsky

http://www.w3.org/2001/XMLSchema#string

Fadi N Salloum

http://www.w3.org/2001/XMLSchema#string

Jeremy Thompson

http://www.w3.org/2001/XMLSchema#string

Jun He

http://www.w3.org/2001/XMLSchema#string

Karol Szczepanek

http://www.w3.org/2001/XMLSchema#string

Qun Chen

http://www.w3.org/2001/XMLSchema#string

Ying Hu

http://www.w3.org/2001/XMLSchema#string

P2860

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

The STAT3 inhibitor stattic impairs cardiomyocyte mitochondrial function through increased reactive oxygen species formation

STATs and gene regulation

Disruption of astrocyte STAT3 signaling decreases mitochondrial function and increases oxidative stress in vitro.

Transient complex I inhibition at the onset of reperfusion by extracellular acidification decreases cardiac injury

Reversible blockade of electron transport with amobarbital at the onset of reperfusion attenuates cardiac injury.

Induction of microRNA-21 with exogenous hydrogen sulfide attenuates myocardial ischemic and inflammatory injury in mice

Leptin-induced cardioprotection involves JAK/STAT signaling that may be linked to the mitochondrial permeability transition pore.

Inhibition of permeability transition pore opening by mitochondrial STAT3 and its role in myocardial ischemia/reperfusion.

What does Stat3 do?

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s00395-015-0509-2

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53

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10.1007/S00395-015-0509-2

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6

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110

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2015-09-11T00:00:00Z

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281681625

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26358226

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reperfusion injury