Genomic island 2 is an unstable genetic element contributing to Brucella lipopolysaccharide spontaneous smooth-to-rough dissociation.
about
Deletion of the GI-2 integrase and the wbkA flanking transposase improves the stability of Brucella melitensis Rev 1 vaccineLipopolysaccharide-Deficient Brucella Variants Arise Spontaneously during Infection.What have we learned from brucellosis in the mouse model?Brucella canis is an intracellular pathogen that induces a lower proinflammatory response than smooth zoonotic counterpartsSmooth to Rough Dissociation in Brucella: The Missing Link to VirulenceStructural Studies of Lipopolysaccharide-defective Mutants from Brucella melitensis Identify a Core Oligosaccharide Critical in Virulence.Diverse genetic regulon of the virulence-associated transcriptional regulator MucR in Brucella abortus 2308.Brucella genomics as we enter the multi-genome era.Internal affairs: investigating the Brucella intracellular lifestyle.Brucella dissociation is essential for macrophage egress and bacterial dissemination.Spontaneous excision of the O-polysaccharide wbkA glycosyltranferase gene is a cause of dissociation of smooth to rough Brucella colonies.
P2860
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P2860
Genomic island 2 is an unstable genetic element contributing to Brucella lipopolysaccharide spontaneous smooth-to-rough dissociation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Genomic island 2 is an unstabl ...... smooth-to-rough dissociation.
@en
type
label
Genomic island 2 is an unstabl ...... smooth-to-rough dissociation.
@en
prefLabel
Genomic island 2 is an unstabl ...... smooth-to-rough dissociation.
@en
P2860
P50
P356
P1476
Genomic island 2 is an unstabl ...... smooth-to-rough dissociation.
@en
P2860
P304
P356
10.1128/JB.00838-10
P577
2010-10-15T00:00:00Z