about
Respiratory syncytial virus infection and G and/or SH protein expression contribute to substance P, which mediates inflammation and enhanced pulmonary disease in BALB/c mice.The role of neurotrophins in allergic bronchial asthma.The G glycoprotein of respiratory syncytial virus depresses respiratory rates through the CX3C motif and substance P.Regulators of G protein signalling: potential targets for treatment of allergic inflammatory diseases such as asthma.In vivo pharmacological evaluation of compound 48/80-induced airways oedema by MRI.Capsaicin-induced mucus secretion in rat airways assessed in vivo and non-invasively by magnetic resonance imaging.Substance P released from intrinsic airway neurons contributes to ozone-enhanced airway hyperresponsiveness in ferret trachea.Substance P-induced airway hyperreactivity is mediated by neuronal M(2) receptor dysfunction.
P2860
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P2860
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Role of tachykinins in bronchial hyper-responsiveness.
@en
type
label
Role of tachykinins in bronchial hyper-responsiveness.
@en
prefLabel
Role of tachykinins in bronchial hyper-responsiveness.
@en
P2093
P2860
P1476
Role of tachykinins in bronchial hyper-responsiveness.
@en
P2093
Reynolds PN
Scicchitano R
P2860
P304
P356
10.1111/J.1440-1681.1997.TB01819.X
P577
1997-03-01T00:00:00Z