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SAR Studies of Exosite-Binding Substrate-Selective Inhibitors of A Disintegrin And Metalloprotease 17 (ADAM17) and Application as Selective in Vitro Probes.Discovery of an enzyme and substrate selective inhibitor of ADAM10 using an exosite-binding glycosylated substrateEmbryonic stem cell-derived M2-like macrophages delay cutaneous wound healing.CX3CR1 is a gatekeeper for intestinal barrier integrity in mice: Limiting steatohepatitis by maintaining intestinal homeostasis.The DRF motif of CXCR6 as chemokine receptor adaptation to adhesion.The perioperative time course and clinical significance of the chemokine CXCL16 in patients undergoing cardiac surgeryA novel peptide ADAM8 inhibitor attenuates bronchial hyperresponsiveness and Th2 cytokine mediated inflammation of murine asthmatic models.The role of ADAM-mediated shedding in vascular biology.A cytoplasmic C-terminal fragment of Syndecan-1 is generated by sequential proteolysis and antagonizes Syndecan-1 dependent lung tumor cell migration.ADAM-family metalloproteinases in lung inflammation: potential therapeutic targets.Systematic substrate identification indicates a central role for the metalloprotease ADAM10 in axon targeting and synapse functionLeukocytes require ADAM10 but not ADAM17 for their migration and inflammatory recruitment into the alveolar space.Fine Tuning Cell Migration by a Disintegrin and Metalloproteinases.Stimulated release and functional activity of surface expressed metalloproteinase ADAM17 in exosomes.Requirements for leukocyte transmigration via the transmembrane chemokine CX3CL1.Distinct role of the intracellular C-terminus for subcellular expression, shedding and function of the murine transmembrane chemokine CX3CL1.PP020. Evidence of a preventive role of Nrf2 in preeclampsia.Myeloid A disintegrin and metalloproteinase domain 10 deficiency modulates atherosclerotic plaque composition by shifting the balance from inflammation toward fibrosis.Lung endothelial ADAM17 regulates the acute inflammatory response to lipopolysaccharideShear Stress Counteracts Endothelial CX3CL1 Induction and Monocytic Cell Adhesion.Smooth muscle cells relay acute pulmonary inflammation via distinct ADAM17/ErbB axes.Illumination is necessary and sufficient to induce histone acetylation independent of transcriptional activity at the C4-specific phosphoenolpyruvate carboxylase promoter in maize.Considerations on inhibition approaches for proinflammatory functions of ADAM proteases.A transmembrane C-terminal fragment of syndecan-1 is generated by the metalloproteinase ADAM17 and promotes lung epithelial tumor cell migration and lung metastasis formation
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description
hulumtuese
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researcher
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ricercatrice
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wetenschapper
@nl
հետազոտող
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name
Daniela Dreymueller
@ast
Daniela Dreymueller
@en
Daniela Dreymueller
@es
Daniela Dreymueller
@fr
Daniela Dreymueller
@nl
Daniela Dreymueller
@sl
type
label
Daniela Dreymueller
@ast
Daniela Dreymueller
@en
Daniela Dreymueller
@es
Daniela Dreymueller
@fr
Daniela Dreymueller
@nl
Daniela Dreymueller
@sl
prefLabel
Daniela Dreymueller
@ast
Daniela Dreymueller
@en
Daniela Dreymueller
@es
Daniela Dreymueller
@fr
Daniela Dreymueller
@nl
Daniela Dreymueller
@sl
P214
P1053
B-9590-2016
P106
P21
P213
0000 0000 7003 8259
P214
P31
P3829
P496
0000-0002-9702-8581
P735
P7859
viaf-95431948