Colonic epithelial physiology is altered in response to the bacterial superantigen Yersinia pseudotuberculosis mitogen.
about
Superantigen YPMa exacerbates the virulence of Yersinia pseudotuberculosis in mice.Loss of NHE3 alters gut microbiota composition and influences Bacteroides thetaiotaomicron growth.Superantigen immune stimulation activates epithelial STAT-1 and PI 3-K: PI 3-K regulation of permeability.Interleukins 4 and 13 increase intestinal epithelial permeability by a phosphatidylinositol 3-kinase pathway. Lack of evidence for STAT 6 involvement.Dextran sulfate sodium-induced colonic histopathology, but not altered epithelial ion transport, is reduced by inhibition of phosphodiesterase activity.
P2860
Colonic epithelial physiology is altered in response to the bacterial superantigen Yersinia pseudotuberculosis mitogen.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Colonic epithelial physiology ...... ia pseudotuberculosis mitogen.
@en
type
label
Colonic epithelial physiology ...... ia pseudotuberculosis mitogen.
@en
prefLabel
Colonic epithelial physiology ...... ia pseudotuberculosis mitogen.
@en
P2093
P356
P1476
Colonic epithelial physiology ...... ia pseudotuberculosis mitogen.
@en
P2093
P304
P356
10.1086/315075
P407
P577
1999-11-01T00:00:00Z