The yopJ locus is required for Yersinia-mediated inhibition of NF-kappaB activation and cytokine expression: YopJ contains a eukaryotic SH2-like domain that is essential for its repressive activity.
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The apoptotic signaling pathway activated by Toll-like receptor-2The Yersinia YopE and YopH type III effector proteins enhance bacterial proliferation following contact with eukaryotic cellsYersinia type III effectors perturb host innate immune responsesIntegrating high-content imaging and chemical genetics to probe host cellular pathways critical for Yersinia pestis infectionLegionella pneumophila type II secretion dampens the cytokine response of infected macrophages and epitheliaPseudomonas aeruginosa induces type-III-secretion-mediated apoptosis of macrophages and epithelial cells.Yersinia has a tropism for B and T cell zones of lymph nodes that is independent of the type III secretion system.Yersinia enterocolitica YopP inhibits MAP kinase-mediated antigen uptake in dendritic cells.Macrophage activation redirects yersinia-infected host cell death from apoptosis to caspase-1-dependent pyroptosis.Reduced secretion of YopJ by Yersinia limits in vivo cell death but enhances bacterial virulence.Cycle inhibiting factors (CIFs) are a growing family of functional cyclomodulins present in invertebrate and mammal bacterial pathogensYersinia pestis endowed with increased cytotoxicity is avirulent in a bubonic plague model and induces rapid protection against pneumonic plague.The tranquilizing injection of Yersinia proteins: a pathogen's strategy to resist host defense.The Yersinia deadly kiss.Diverse agents act at multiple levels to inhibit the Rel/NF-kappaB signal transduction pathway.GAP activity of the Yersinia YopE cytotoxin specifically targets the Rho pathway: a mechanism for disruption of actin microfilament structure.Bacterial regulation of intestinal immune responses.Type III secretion: a bacterial device for close combat with cells of their eukaryotic hostMolecular and cell biology aspects of plagueDifferential induction of complement fragment C5a and inflammatory cytokines during intramammary infections with Escherichia coli and Staphylococcus aureus.YopJ of Yersinia spp. is sufficient to cause downregulation of multiple mitogen-activated protein kinases in eukaryotic cellsInvolvement of protein kinase C in Rickettsia rickettsii-induced transcriptional activation of the host endothelial cell.Arginine-143 of Yersinia enterocolitica YopP crucially determines isotype-related NF-kappaB suppression and apoptosis induction in macrophages.NF-kappaB signaling proteins as therapeutic targets for inflammatory bowel diseases.Pathogenic strategies of enteric bacteria.Effect of low- and high-virulence Yersinia enterocolitica strains on the inflammatory response of human umbilical vein endothelial cells.Role of Yops and adhesins in resistance of Yersinia enterocolitica to phagocytosis.Pathogen-induced apoptosis of macrophages: a common end for different pathogenic strategies.Concerted actions of a thermo-labile regulator and a unique intergenic RNA thermosensor control Yersinia virulence.Inhibiting NF-κB activation by small molecules as a therapeutic strategy.Microbiome and immunological interactions.Calcium-regulated type III secretion of Yop proteins by an Escherichia coli hha mutant carrying a Yersinia pestis pCD1 virulence plasmidAcetylation of MEK2 and I kappa B kinase (IKK) activation loop residues by YopJ inhibits signaling.Yersinia effectors target mammalian signalling pathways.Natural selection drives Drosophila immune system evolution.Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions.Yersinia type III secretion: send in the effectors.Streptococcus cristatus attenuates Fusobacterium nucleatum-induced cytokine expression by influencing pathways converging on nuclear factor-κB.The Yersinia Ysc-Yop 'type III' weaponry.
P2860
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P2860
The yopJ locus is required for Yersinia-mediated inhibition of NF-kappaB activation and cytokine expression: YopJ contains a eukaryotic SH2-like domain that is essential for its repressive activity.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
The yopJ locus is required for ...... l for its repressive activity.
@en
type
label
The yopJ locus is required for ...... l for its repressive activity.
@en
prefLabel
The yopJ locus is required for ...... l for its repressive activity.
@en
P2093
P2860
P1476
The yopJ locus is required for ...... l for its repressive activity.
@en
P2093
Dukuzumuremyi JM
Neurath MF
Pettersson S
Schesser K
Wolf-Watz H
P2860
P304
P356
10.1046/J.1365-2958.1998.00851.X
P407
P577
1998-06-01T00:00:00Z