Hepatocyte DACH1 Is Increased in Obesity via Nuclear Exclusion of HDAC4 and Promotes Hepatic Insulin Resistance
about
Calcium signalling and ER stress in insulin resistance and atherosclerosis.Novel biological therapies for the treatment of diabetic foot ulcers.Characterization of the Molecular Mechanisms Underlying Glucose Stimulated Insulin Secretion from Isolated Pancreatic β-cells Using Post-translational Modification Specific Proteomics (PTMomics).CAMKIIγ suppresses an efferocytosis pathway in macrophages and promotes atherosclerotic plaque necrosis.Hepatocyte-secreted DPP4 in obesity promotes adipose inflammation and insulin resistance.Pharmacologic ATF6 activating compounds are metabolically activated to selectively modify endoplasmic reticulum proteins
P2860
Hepatocyte DACH1 Is Increased in Obesity via Nuclear Exclusion of HDAC4 and Promotes Hepatic Insulin Resistance
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Hepatocyte DACH1 Is Increased ...... tes Hepatic Insulin Resistance
@en
type
label
Hepatocyte DACH1 Is Increased ...... tes Hepatic Insulin Resistance
@en
prefLabel
Hepatocyte DACH1 Is Increased ...... tes Hepatic Insulin Resistance
@en
P2093
P2860
P1433
P1476
Hepatocyte DACH1 Is Increased ...... tes Hepatic Insulin Resistance
@en
P2093
Beth Schrope
Devram S Ghorpade
Jane Cristina de Souza
Marc Bessler
Melissa Bagloo
Richard Pestell
P2860
P304
P356
10.1016/J.CELREP.2016.05.006
P50
P577
2016-05-26T00:00:00Z