ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change. - Wikidata - wikimedia - TriplyDB

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

http://www.wikidata.org/entity/Q41502785

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Molecular interaction and functional regulation of ClC-3 by Ca2+/calmodulin-dependent protein kinase II (CaMKII) in human malignant glioma.CFTR-mediated halide transport in phagosomes of human neutrophils.Activation of swelling-activated chloride current by tumor necrosis factor-alpha requires ClC-3-dependent endosomal reactive oxygen production RNA interference against CFTR affects HL60-derived neutrophil microbicidal function.Priming of neutrophils and differentiated PLB-985 cells by pathophysiological concentrations of TNF-α is partially oxygen dependent Novel CLC3 transcript variants in blood eosinophils and increased CLC3 expression in nasal lavage and blood eosinophils of asthmatics.Interferon Regulatory Factor 6 Has a Protective Role in the Host Response to Endotoxic Shock Chloride Channel 3 Channels in the Activation and Migration of Human Blood Eosinophils in Allergic Asthma.The ClC-3 chloride channels in cardiovascular disease Threonine532 phosphorylation in ClC-3 channels is required for angiotensin II-induced Cl(-) current and migration in cultured vascular smooth muscle cells.Ionic currents of human trabecular meshwork cells from control and glaucoma subjects Salt, chloride, bleach, and innate host defense.Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3 Phenomics of cardiac chloride channels: the systematic study of chloride channel function in the heart.Phenomics of cardiac chloride channels.Defining signal transduction by inositol phosphates.Roles of ion transport in control of cell motility.Cell biology and physiology of CLC chloride channels and transporters.Chloride transport in functionally active phagosomes isolated from Human neutrophils.Analysis of electrophysiological properties and responses of neutrophils.Complement C5a-Induced Changes in Neutrophil Morphology During Inflammation.Chloride flux in phagocytes.The calcium-activated potassium channel KCa3.1 plays a central role in the chemotactic response of mammalian neutrophils.Myosin II is essential for the spatiotemporal organization of traction forces during cell motility.4D Tumorigenesis Model for Quantitating Coalescence, Directed Cell Motility and Chemotaxis, Identifying Unique Cell Behaviors, and Testing Anticancer Drugs.Overexpression of chloride channel-3 is associated with the increased migration and invasion ability of ectopic endometrial cells from patients with endometriosis.

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P2860

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

http://www.wikidata.org/entity/Q41502785

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

@en

type

label

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

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prefLabel

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

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P1433

Journal of Biological Chemistry

P1476

ClC-3 and IClswell are required for normal neutrophil chemotaxis and shape change.

@en

P2093

A Paige Davis Volk

http://www.w3.org/2001/XMLSchema#string

Christine K Heise

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Christopher M Artman

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David R Soll

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Deborah Wessels

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Fred S Lamb

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Jami L Hougen

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Jessica G Moreland

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Kenneth A Volk

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William M Nauseef

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P2860

Disruption of ClC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus

Regulation of human CLC-3 channels by multifunctional Ca2+/calmodulin-dependent protein kinase

ClC-3 is a fundamental molecular component of volume-sensitive outwardly rectifying Cl- channels and volume regulation in HeLa cells and Xenopus laevis oocytes

The chemotaxis defect of Shwachman-Diamond Syndrome leukocytes

Anion channels, including ClC-3, are required for normal neutrophil oxidative function, phagocytosis, and transendothelial migration

Cytokine activation of nuclear factor kappa B in vascular smooth muscle cells requires signaling endosomes containing Nox1 and ClC-3

Of mice and not men: differences between mouse and human immunology

The N-formylpeptide receptor (FPR) and a second G(i)-coupled receptor mediate fMet-Leu-Phe-stimulated activation of NADPH oxidase in murine neutrophils

Ability of polymorphonuclear leukocytes to orient in gradients of chemotactic factors

Function and spatial distribution of ion channels and transporters in cell migration.

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34315-34326

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scholarly article

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10.1074/JBC.M803141200

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49

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283

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2008-10-07T00:00:00Z

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18840613

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2590705

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