The Ca2+ influx through the mammalian skeletal muscle dihydropyridine receptor is irrelevant for muscle performance.
about
Voltage modulates halothane-triggered Ca2+ release in malignant hyperthermia-susceptible muscle.De novo reconstitution reveals the proteins required for skeletal muscle voltage-induced Ca2+ release.A skeletal muscle L-type Ca2+ channel with a mutation in the selectivity filter (CaV1.1 E1014K) conducts K.When muscle Ca2+ channels carry monovalent cations through gating pores: insights into the pathophysiology of type 1 hypokalaemic periodic paralysis.Distinct transcriptomic changes in E14.5 mouse skeletal muscle lacking RYR1 or Cav1.1 converge at E18.5.
P2860
The Ca2+ influx through the mammalian skeletal muscle dihydropyridine receptor is irrelevant for muscle performance.
description
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name
The Ca2+ influx through the ma ...... levant for muscle performance.
@en
type
label
The Ca2+ influx through the ma ...... levant for muscle performance.
@en
prefLabel
The Ca2+ influx through the ma ...... levant for muscle performance.
@en
P2093
P2860
P1476
The Ca2+ influx through the ma ...... elevant for muscle performance
@en
P2093
Kai Schrötter
Manfred Grabner
Werner Melzer
P2860
P2888
P356
10.1038/S41467-017-00629-X
P407
P577
2017-09-07T00:00:00Z