about
NFI transcription factors interact with FOXA1 to regulate prostate-specific gene expression.Mouse models of prostate cancer: picking the best model for the question.Soluble E-cadherin: more than a symptom of disease.Loss of FOXA1 Drives Sexually Dimorphic Changes in Urothelial Differentiation and Is an Independent Predictor of Poor Prognosis in Bladder Cancer.NF-κB and androgen receptor variant expression correlate with human BPH progressionNfib Regulates Transcriptional Networks That Control the Development of Prostatic HyperplasiaNF-κB and androgen receptor variant 7 induce expression of SRD5A isoforms and confer 5ARI resistance.Activation of GRP/GRP-R signaling contributes to castration-resistant prostate cancer progression.Androgen receptor differentially regulates the proliferation of prostatic epithelial cells in vitro and in vivo.EGF promotes the shedding of soluble E-cadherin in an ADAM10-dependent manner in prostate epithelial cells.FOXA1 deletion in luminal epithelium causes prostatic hyperplasia and alteration of differentiated phenotypeSOX2 expression in the developing, adult, as well as, diseased prostateTherapy-induced small-cell disease: from mouse to man and backProstatic osteopontin expression is associated with symptomatic benign prostatic hyperplasia
P50
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P50
description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Magdalena M Grabowska
@ast
Magdalena M Grabowska
@en
Magdalena M Grabowska
@es
Magdalena M Grabowska
@sl
type
label
Magdalena M Grabowska
@ast
Magdalena M Grabowska
@en
Magdalena M Grabowska
@es
Magdalena M Grabowska
@sl
prefLabel
Magdalena M Grabowska
@ast
Magdalena M Grabowska
@en
Magdalena M Grabowska
@es
Magdalena M Grabowska
@sl
P106
P1960
arB_ZQEAAAAJ
P21
P31
P496
0000-0003-1550-4925