TM6SF2 E167K variant is associated with severe steatosis in chronic hepatitis C, regardless of PNPLA3 polymorphism. - Wikidata - wikimedia - TriplyDB

TM6SF2 E167K variant is associated with severe steatosis in chronic hepatitis C, regardless of PNPLA3 polymorphism.

TM6SF2 E167K variant is associated with severe steatosis in chronic hepatitis C, regardless of PNPLA3 polymorphism.

http://www.wikidata.org/entity/Q41549262

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NAFLD and NASH in HCV Infection: Prevalence and Significance in Hepatic and Extrahepatic Manifestations TM6SF2 E167K Variant, a Novel Genetic Susceptibility Variant, Contributing to Nonalcoholic Fatty Liver Disease Hepatitis C virus markers in infection by hepatitis C virus: In the era of directly acting antivirals Role of occult hepatitis B virus infection in chronic hepatitis C Lowered expression of microRNA-125a-5p in human hepatocellular carcinoma and up-regulation of its oncogenic targets sirtuin-7, matrix metalloproteinase-11, and c-Raf Hepatic Transmembrane 6 Superfamily Member 2 Regulates Cholesterol Metabolism in Mice Meta-analysis of the influence of TM6SF2 E167K variant on Plasma Concentration of Aminotransferases across different Populations and Diverse Liver Phenotypes.Insights from Genome-Wide Association Analyses of Nonalcoholic Fatty Liver Disease.Hepatitis B virus infection in undocumented immigrants and refugees in Southern Italy: demographic, virological, and clinical features.Occult HBV infection in HCC and cirrhotic tissue of HBsAg-negative patients: a virological and clinical study.Novel association of TM6SF2 rs58542926 genotype with increased serum tyrosine levels and decreased apoB-100 particles in Finns.Clinical impact of the hepatitis C virus mutations in the era of directly acting antivirals.Hepatic steatosis in HCV-infected persons in the direct-acting antiviral era.The effect of the TM6SF2 E167K variant on liver steatosis and fibrosis in patients with chronic hepatitis C: a meta-analysis.TM6SF2 E167K variant predicts severe liver fibrosis for human immunodeficiency/hepatitis C virus co-infected patients, and severe steatosis only for a non-3 hepatitis C virus genotype.Transmembrane 6 superfamily member 2 gene E167K variant impacts on steatosis and liver damage in chronic hepatitis C patients.TM6SF2 rs58542926 is not associated with steatosis and fibrosis in large cohort of patients with genotype 1 chronic hepatitis C.Age might confound the impact of the transmembrane 6 superfamily member 2 E167K polymorphism on hepatic fibrosis in hepatitis C virus-infected patients.The rs738409 polymorphism of the PNPLA3 gene is associated with hepatic steatosis and fibrosis in Brazilian patients with chronic hepatitis C.The Genetics of Clinical Liver Diseases: Insight into the E167K Variant A systematic review of the present and future of non-alcoholic fatty liver disease

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TM6SF2 E167K variant is associated with severe steatosis in chronic hepatitis C, regardless of PNPLA3 polymorphism.

http://www.wikidata.org/entity/Q41549262

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2015 nî lūn-bûn

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Adriana Boemio

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Aldo Marrone

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Emanuele Miraglia del Giudice

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Grazia Cirillo

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Luigi E Adinolfi

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Zampino Rosa

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P2860

TM6SF2 is a regulator of liver fat metabolism influencing triglyceride secretion and hepatic lipid droplet content

Common variants at 30 loci contribute to polygenic dyslipidemia

Cannabinoid receptor 2-63 QQ variant is associated with persistently normal aminotransferase serum levels in chronic hepatitis C

Design and validation of a histological scoring system for nonalcoholic fatty liver disease

Histological grading and staging of chronic hepatitis

Genome-wide association analysis identifies variants associated with nonalcoholic fatty liver disease that have distinct effects on metabolic traits.

EASL Clinical Practice Guidelines: management of hepatitis C virus infection.

Systematic evaluation of coding variation identifies a candidate causal variant in TM6SF2 influencing total cholesterol and myocardial infarction risk

Exome-wide association study identifies a TM6SF2 variant that confers susceptibility to nonalcoholic fatty liver disease.

Disease progression in chronic hepatitis C: modifiable and nonmodifiable factors.

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10.1111/LIV.12781

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8

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35

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Maria Stanzione

Evangelista Sagnelli

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270598067

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