Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition. - Wikidata - wikimedia - TriplyDB

Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition.

Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition.

http://www.wikidata.org/entity/Q41584964

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Endothelial-Mesenchymal Transition in Regenerative Medicine Dipeptidyl peptidase-4 and kidney fibrosis in diabetes DNMT1 and HDAC2 Cooperate to Facilitate Aberrant Promoter Methylation in Inorganic Phosphate-Induced Endothelial-Mesenchymal Transition.Serine Protease Activation Essential for Endothelial-Mesenchymal Transition in Vascular Calcification.A mouse model of endocardial fibroelastosis Pigment epithelium-derived factor attenuates myocardial fibrosis via inhibiting Endothelial-to-Mesenchymal Transition in rats with acute myocardial infarction.The third path of tubulointerstitial fibrosis: aberrant endothelial secretome.Macrophage-derived foam cells impair endothelial barrier function by inducing endothelial-mesenchymal transition via CCL-4.The future of pleiotropic therapy in heart failure. Lessons from the benefits of exercise training on endothelial function.Stress-Induced Premature Senescence of Endothelial and Endothelial Progenitor Cells.DNA methylation regulated gene expression in organ fibrosis.Re-evaluation of hypoplastic left heart syndrome from a developmental and morphological perspective.Fibroblasts in an endocardial fibroelastosis disease model mainly originate from mesenchymal derivatives of epicardium.Endocardial Fibroelastosis is Secondary to Hemodynamic Alterations in the Chick Embryonic Model of Hypoplastic Left Heart Syndrome.Transcriptional regulation of endothelial-to-mesenchymal transition in cardiac fibrosis: role of myocardin-related transcription factor A and activating transcription factor 3.Hypoxia-induced endothelial-mesenchymal transition is associated with RASAL1 promoter hypermethylation in human coronary endothelial cells.Endothelial to Mesenchymal Transition Represents a Key Link in the Interaction between Inflammation and Endothelial Dysfunction.High-fidelity CRISPR/Cas9- based gene-specific hydroxymethylation rescues gene expression and attenuates renal fibrosis

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Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition.

http://www.wikidata.org/entity/Q41584964

description

2015 nî lūn-bûn

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2015年の論文

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name

Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition.

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type

label

Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition.

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prefLabel

Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition.

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CIRCRESAHA.116.305629

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Circulation Research

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Endocardial fibroelastosis is caused by aberrant endothelial to mesenchymal transition

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Elisabeth M Zeisberg

http://www.w3.org/2001/XMLSchema#string

Fouzi Alnour

http://www.w3.org/2001/XMLSchema#string

Ingeborg Friehs

http://www.w3.org/2001/XMLSchema#string

Ivan Melnychenko

http://www.w3.org/2001/XMLSchema#string

Maria Iascone

http://www.w3.org/2001/XMLSchema#string

Michael Zeisberg

http://www.w3.org/2001/XMLSchema#string

Pedro J Del Nido

http://www.w3.org/2001/XMLSchema#string

Raghu Kalluri

http://www.w3.org/2001/XMLSchema#string

Tachi Zhong Hu

http://www.w3.org/2001/XMLSchema#string

Xingbo Xu

http://www.w3.org/2001/XMLSchema#string

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Identification of de novo mutations and rare variants in hypoplastic left heart syndrome

Nebulette mutations are associated with dilated cardiomyopathy and endocardial fibroelastosis

Development of heart valves requires Gata4 expression in endothelial-derived cells

TGF-beta signalling from cell membrane to nucleus through SMAD proteins

Smads: transcriptional activators of TGF-beta responses

Viral infection of the myocardium in endocardial fibroelastosis. Molecular evidence for the role of mumps virus as an etiologic agent

Early embryonic lethality in Bmp5;Bmp7 double mutant mice suggests functional redundancy within the 60A subgroup

Connexin43 knockdown accelerates wound healing but inhibits mesenchymal transition after corneal endothelial injury in vivo

TGF-β-induced mesenchymal transition of MS-1 endothelial cells requires Smad-dependent cooperative activation of Rho signals and MRTF-A

How cells read TGF-beta signals

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857-866

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scholarly article

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10.1161/CIRCRESAHA.116.305629

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5

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116

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2015-01-13T00:00:00Z

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25587097

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Collaborative Research Centre (CRC) 1002: "Modulatory Units in Heart Failure"

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4344885

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