Hypoxia is an effective stimulus for vesicular release of ATP from human umbilical vein endothelial cells. - Wikidata - wikimedia - TriplyDB

Hypoxia is an effective stimulus for vesicular release of ATP from human umbilical vein endothelial cells.

Hypoxia is an effective stimulus for vesicular release of ATP from human umbilical vein endothelial cells.

http://www.wikidata.org/entity/Q41589244

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Immunologic Consequences of Hypoxia during Critical Illness.Interactions between local dilator and sympathetic vasoconstrictor influences in skeletal muscle in acute and chronic hypoxia.Expression of P2 Purinergic Receptors in Mesenchymal Stem Cells and Their Roles in Extracellular Nucleotide Regulation of Cell Functions.Hypoxia in Atherogenesis.Endothelial-smooth muscle cell interactions in the regulation of vascular tone in skeletal muscle.Adenosine triphosphate is a critical determinant for VEGFR signal during hypoxia.Role of thiosulfate in hydrogen sulfide-dependent redox signaling in endothelial cells.Pharmacological dissection of the cellular mechanisms associated to the spontaneous and the mechanically stimulated ATP release by mesentery endothelial cells: roles of thrombin and TRPV.Atheroprone flow activates inflammation via endothelial ATP-dependent P2X7-p38 signalling.Mechanisms of ATP Release by Inflammatory Cells.Mitochondrial targeting as a novel therapy for stroke

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Hypoxia is an effective stimulus for vesicular release of ATP from human umbilical vein endothelial cells.

http://www.wikidata.org/entity/Q41589244

description

2015 nî lūn-bûn

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2015年の論文

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J.PLACENTA.2015.04.005

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J M Marshall

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W K Lim To

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Brefeldin A: insights into the control of membrane traffic and organelle structure

Involvement of Rho-kinase and tyrosine kinase in hypotonic stress-induced ATP release in bovine aortic endothelial cells

A specific inhibitor of phosphatidylinositol 3-kinase, 2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one (LY294002)

Alteration of intracellular traffic by monensin; mechanism, specificity and relationship to toxicity

Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria

S-nitrosylation inhibits pannexin 1 channel function

Mechanisms of ATP release and signalling in the blood vessel wall.

ATP release from vascular endothelia occurs across Cx43 hemichannels and is attenuated during hypoxia.

Fetal anaemia and its relation with increased concentrations of adenosine

Initiation of purinergic signaling by exocytosis of ATP-containing vesicles in liver epithelium

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