MEK5/ERK5 signaling inhibition increases colon cancer cell sensitivity to 5-fluorouracil through a p53-dependent mechanism.
about
The role of GLI1 for 5-Fu resistance in colorectal cancerChemoresistance of colorectal cancer to 5-fluorouracil is associated with silencing of the BNIP3 gene through aberrant methylationOncogenic signaling of MEK5-ERK5.JNK, p38, ERK, and SGK1 Inhibitors in Cancer.Changes in metabolism affect expression of ABC transporters through ERK5 and depending on p53 status.Acriflavine enhances the antitumor activity of the chemotherapeutic drug 5-fluorouracil in colorectal cancer cells.
P2860
MEK5/ERK5 signaling inhibition increases colon cancer cell sensitivity to 5-fluorouracil through a p53-dependent mechanism.
description
2016 nî lūn-bûn
@nan
2016年の論文
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2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
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2016年论文
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name
MEK5/ERK5 signaling inhibition ...... ugh a p53-dependent mechanism.
@en
type
label
MEK5/ERK5 signaling inhibition ...... ugh a p53-dependent mechanism.
@en
prefLabel
MEK5/ERK5 signaling inhibition ...... ugh a p53-dependent mechanism.
@en
P2860
P50
P356
P1433
P1476
MEK5/ERK5 signaling inhibition ...... ugh a p53-dependent mechanism.
@en
P2093
Pedro M Borralho
P2860
P304
34322-34340
P356
10.18632/ONCOTARGET.9107
P407
P577
2016-04-29T00:00:00Z