Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

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Cleavage of Fibulin-2 by the aggrecanases ADAMTS-4 and ADAMTS-5 contributes to the tumorigenic potential of breast cancer cells.Stromal Versican Regulates Tumor Growth by Promoting Angiogenesis.Evaluation of Tumor Vasculature Using a Syngeneic Tumor Model in Wild-Type and Genetically Modified Mice.

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P2860

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

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http://www.wikidata.org/entity/Q41610199

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

@en

type

Item

label

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

@en

prefLabel

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

@en

P1476

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

@en

P2093

Estefanía Martino-Echarri

http://www.w3.org/2001/XMLSchema#string

María Del Carmen Plaza-Calonge

http://www.w3.org/2001/XMLSchema#string

Rubén Fernández-Rodríguez

http://www.w3.org/2001/XMLSchema#string

P2860

METH-1, a human ortholog of ADAMTS-1, and METH-2 are members of a new family of proteins with angio-inhibitory activity

ADAMTS1 cleaves aggrecan at multiple sites and is differentially inhibited by metalloproteinase inhibitors

Versican V1 proteolysis in human aorta in vivo occurs at the Glu441-Ala442 bond, a site that is cleaved by recombinant ADAMTS-1 and ADAMTS-4

High cancer cell death in syngeneic tumors developed in host mice deficient for the stromelysin-3 matrix metalloproteinase

Adamts-1 is essential for the development and function of the urogenital system

Toward understanding and exploiting tumor heterogeneity

Breast cancer cells induce stromal fibroblasts to secrete ADAMTS1 for cancer invasion through an epigenetic change

ADAMTS1 interacts with, cleaves, and modifies the extracellular location of the matrix inhibitor tissue factor pathway inhibitor-2.

The cleavage of semaphorin 3C induced by ADAMTS1 promotes cell migration

ADAMTS1 alters blood vessel morphology and TSP1 levels in LNCaP and LNCaP-19 prostate tumors

P304

34507-34519

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

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10.18632/ONCOTARGET.8922

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Juan Carlos Rodriguez-manzaneque

Carlos Peris-Torres

F J Rodríguez-Baena

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2016-04-22T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27120788

http://www.w3.org/2001/XMLSchema#string

P932

5085172

http://www.w3.org/2001/XMLSchema#string

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

about

P2860

P2860

Stroma-derived but not tumor ADAMTS1 is a main driver of tumor growth and metastasis.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932