Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons.
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Mutation of SOD1 in ALS: a gain of a loss of functionCLUH regulates mitochondrial biogenesis by binding mRNAs of nuclear-encoded mitochondrial proteinsThe zinc finger protein ZPR1 is a potential modifier of spinal muscular atrophy.Rnf165/Ark2C enhances BMP-Smad signaling to mediate motor axon extensionZPR1 is essential for survival and is required for localization of the survival motor neurons (SMN) protein to Cajal bodies.Neuroprotective effect of Nrf2/ARE activators, CDDO ethylamide and CDDO trifluoroethylamide, in a mouse model of amyotrophic lateral sclerosisNAIP interacts with hippocalcin and protects neurons against calcium-induced cell death through caspase-3-dependent and -independent pathwaysGranulocyte-colony stimulating factor improves outcome in a mouse model of amyotrophic lateral sclerosisAn RNA polymerase II construct synthesizes short-hairpin RNA with a quantitative indicator and mediates highly efficient RNAi3D in vitro modeling of the central nervous systemNeuronal-specific deficiency of the splicing factor Tra2b causes apoptosis in neurogenic areas of the developing mouse brainAmyotrophic lateral sclerosis-linked mutant VAPB inclusions do not interfere with protein degradation pathways or intracellular transport in a cultured cell modelThe spinal muscular atrophy disease protein SMN is linked to the Golgi networkA loss of FUS/TLS function leads to impaired cellular proliferationAn in vivo reporter system for measuring increased inclusion of exon 7 in SMN2 mRNA: potential therapy of SMAAlsin, the product of ALS2 gene, suppresses SOD1 mutant neurotoxicity through RhoGEF domain by interacting with SOD1 mutantsExendin-4 ameliorates motor neuron degeneration in cellular and animal models of amyotrophic lateral sclerosisThe LIM/homeodomain protein Islet1 recruits Janus tyrosine kinases and signal transducer and activator of transcription 3 and stimulates their activitiesFunctional inactivation of the genome-wide association study obesity gene neuronal growth regulator 1 in mice causes a body mass phenotypeMammalian Gup1, a homolog of Saccharomyces cerevisiae glycerol uptake/transporter 1, acts as a negative regulator for N-terminal palmitoylation of Sonic hedgehogDeficiency of the zinc finger protein ZPR1 causes neurodegenerationZonisamide Enhances Neurite Elongation of Primary Motor Neurons and Facilitates Peripheral Nerve Regeneration In Vitro and in a Mouse ModelSuppression of p75 neurotrophin receptor surface expression with intrabodies influences Bcl-xL mRNA expression and neurite outgrowth in PC12 cellsDynamic role of kallikrein 6 in traumatic spinal cord injuryADP-ribosylhydrolase activity of Chikungunya virus macrodomain is critical for virus replication and virulenceSuppressing mutation-induced protein aggregation in mammalian cells by mutating residues significantly displaced upon the original mutationImportant modifications by sugammadex, a modified γ-cyclodextrin, of ion currents in differentiated NSC-34 neuronal cellsCis-suppression to arrest protein aggregation in mammalian cells.A revisited folding reporter for quantitative assay of protein misfolding and aggregation in mammalian cells.Palmitoylation of superoxide dismutase 1 (SOD1) is increased for familial amyotrophic lateral sclerosis-linked SOD1 mutantsS-nitrosothiol depletion in amyotrophic lateral sclerosis.Androgen regulation of axon growth and neurite extension in motoneuronsViral delivery of antioxidant genes as a therapeutic strategy in experimental models of amyotrophic lateral sclerosis.FUS/TLS assembles into stress granules and is a prosurvival factor during hyperosmolar stressRabies virus envelope glycoprotein targets lentiviral vectors to the axonal retrograde pathway in motor neurons.Distinct partitioning of ALS associated TDP-43, FUS and SOD1 mutants into cellular inclusionsBioinformatics Data Mining Approach Suggests Coexpression of AGTPBP1 with an ALS-linked Gene C9orf72.c-Abl inhibition delays motor neuron degeneration in the G93A mouse, an animal model of amyotrophic lateral sclerosis.Analysis of the cytosolic proteome in a cell culture model of familial amyotrophic lateral sclerosis reveals alterations to the proteasome, antioxidant defenses, and nitric oxide synthetic pathways.Mitochondrial proteomic analysis of a cell line model of familial amyotrophic lateral sclerosis.
P2860
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P2860
Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons.
@en
type
label
Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons.
@en
prefLabel
Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons.
@en
P2093
P356
P1476
Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons.
@en
P2093
Blusztajn JK
Cashman NR
Dahrouge S
P304
P356
10.1002/AJA.1001940306
P577
1992-07-01T00:00:00Z