Bromide supplementation exacerbated the renal dysfunction, injury and fibrosis in a mouse model of Alport syndrome.

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Bromide supplementation exacerbated the renal dysfunction, injury and fibrosis in a mouse model of Alport syndrome.

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2017 nî lūn-bûn

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2017年学术文章

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Bromide supplementation exacer ...... ouse model of Alport syndrome.

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Bromide supplementation exacer ...... ouse model of Alport syndrome.

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Bromide supplementation exacer ...... ouse model of Alport syndrome.

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Bromide supplementation exacer ...... ouse model of Alport syndrome.

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Haruka Kojima

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Hirofumi Kai

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Jun Kuwazuru

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Keisuke Teramoto

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Kohei Omachi

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Mary Ann Suico

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Misato Kamura

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Shota Kaseda

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Tsubasa Yokota

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Tsuyoshi Shuto

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P2860

Peroxidasin is secreted and incorporated into the extracellular matrix of myofibroblasts and fibrotic kidney

Bromine is an essential trace element for assembly of collagen IV scaffolds in tissue development and architecture

Role of the TGF-β/BMP-7/Smad pathways in renal diseases

Peroxidasin forms sulfilimine chemical bonds using hypohalous acids in tissue genesis.

The C. elegans peroxidasin PXN-2 is essential for embryonic morphogenesis and inhibits adult axon regeneration.

Mild electrical stimulation and heat shock ameliorates progressive proteinuria and renal inflammation in mouse model of Alport syndrome

Iron chelation by deferoxamine prevents renal interstitial fibrosis in mice with unilateral ureteral obstruction

Alport's syndrome, Goodpasture's syndrome, and type IV collagen.

Metabolism of bromide and its interference with the metabolism of iodine.

Bone morphogenetic protein 7: a novel treatment for chronic renal and bone disease.

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e0183959

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scholarly article

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10.1371/JOURNAL.PONE.0183959

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English

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P478

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2017-09-05T00:00:00Z

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28873450

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5584969

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