Molecular mechanism of miR-181b in heart disease due to pregnancy-induced hypertension syndrome.

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Molecular mechanism of miR-181b in heart disease due to pregnancy-induced hypertension syndrome.

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http://www.wikidata.org/entity/Q41624580

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Molecular mechanism of miR-181 ...... induced hypertension syndrome.

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type

Item

label

Molecular mechanism of miR-181 ...... induced hypertension syndrome.

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prefLabel

Molecular mechanism of miR-181 ...... induced hypertension syndrome.

@en

P1476

Molecular mechanism of miR-181 ...... induced hypertension syndrome.

@en

P2093

Fengyong Yang

http://www.w3.org/2001/XMLSchema#string

Jin Qu

http://www.w3.org/2001/XMLSchema#string

Jinyun Wang

http://www.w3.org/2001/XMLSchema#string

Li Wang

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Zheng Gao

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P2860

Calcium supplementation during pregnancy for preventing hypertensive disorders and related problems

Bioactive factors in uteroplacental and systemic circulation link placental ischemia to generalized vascular dysfunction in hypertensive pregnancy and preeclampsia

How matrix metalloproteinases regulate cell behavior

MicroRNAs regulating meis1 expression and inducing cardiomyocyte proliferation

miR-597 inhibits breast cancer cell proliferation, migration and invasion through FOSL2.

Hypertensive disorders of pregnancy: a systematic review of international clinical practice guidelines.

The association between hypertensive disorders in pregnancy and bronchopulmonary dysplasia: a systematic review.

Tissue inhibitor of metalloproteinase-3 (TIMP3) promotes endothelial apoptosis via a caspase-independent mechanism.

MicroRNA-150 protects the mouse heart from ischaemic injury by regulating cell death.

Placental vascularization alterations in hypertensive disorders complicating pregnancy (HDCP) and small for gestational age with HDCP using three-dimensional power doppler in a prospective case control study.

P304

2953-2959

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P31

scholarly article

P356

10.3892/ETM.2017.4882

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-08-03T00:00:00Z

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P698

28912851

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P932

5585881

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