Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

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NADPH Oxidases and Mitochondria in Vascular Senescence.

P2860

Q55334122-7AE18A89-ECEB-4A14-AC9B-5F0BC92E2B29

P2860

Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

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http://www.wikidata.org/entity/Q41628898

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2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

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type

Item

label

Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

@en

prefLabel

Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

@en

P1476

Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

@en

P2093

Hye Jin Jee

http://www.w3.org/2001/XMLSchema#string

Jeanho Yun

http://www.w3.org/2001/XMLSchema#string

Jee-Hyun Um

http://www.w3.org/2001/XMLSchema#string

Sun Sik Bae

http://www.w3.org/2001/XMLSchema#string

Young Mi Kim

http://www.w3.org/2001/XMLSchema#string

Young Yeon Kim

http://www.w3.org/2001/XMLSchema#string

P2860

Selective activation of Akt1 by mammalian target of rapamycin complex 2 regulates cancer cell migration, invasion, and metastasis

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

The first 30 years of p53: growing ever more complex

NF-kappaB activation by tumour necrosis factor requires the Akt serine-threonine kinase

Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin

DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescence

Mitochondria, oxidants, and aging

mTOR inhibition induces upstream receptor tyrosine kinase signaling and activates Akt

Inhibition of mTORC1 leads to MAPK pathway activation through a PI3K-dependent feedback loop in human cancer

P304

1094-1103

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scholarly article

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10.1111/ACEL.12639

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P478

http://www.w3.org/2001/XMLSchema#string

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2017-07-09T00:00:00Z

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28691365

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P932

5595696

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Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

about

P2860

P2860

Cooperation between p21 and Akt is required for p53-dependent cellular senescence.

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P1433

P1476

P2093

P2860

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P478

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P932