Disrupted nitric oxide signaling due to GUCY1A3 mutations increases risk for moyamoya disease, achalasia and hypertension.
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Identification of novel S-nitrosation sites in soluble guanylyl cyclase, the nitric oxide receptorThiol-Based Redox Modulation of Soluble Guanylyl Cyclase, the Nitric Oxide Receptor.Hydrogen sulphide as a signalling molecule regulating physiopathological processes in gastrointestinal motility.Meta-Analysis of Tourette Syndrome and Attention Deficit Hyperactivity Disorder Provides Support for a Shared Genetic Basis.Stimulators of the soluble guanylyl cyclase: promising functional insights from rare coding atherosclerosis-related GUCY1A3 variants.Structure/function of the soluble guanylyl cyclase catalytic domain.
P2860
Disrupted nitric oxide signaling due to GUCY1A3 mutations increases risk for moyamoya disease, achalasia and hypertension.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Disrupted nitric oxide signali ...... e, achalasia and hypertension.
@en
type
label
Disrupted nitric oxide signali ...... e, achalasia and hypertension.
@en
prefLabel
Disrupted nitric oxide signali ...... e, achalasia and hypertension.
@en
P2093
P2860
P50
P356
P1433
P1476
Disrupted nitric oxide signali ...... se, achalasia and hypertension
@en
P2093
D A Nickerson
D M Milewicz
D Saunders
E Regalado
L Mellor-Crummey
N Hanchard
P2860
P304
P356
10.1111/CGE.12739
P577
2016-02-18T00:00:00Z