about
STIM1 is essential for Fcgamma receptor activation and autoimmune inflammation.Cooperative and alternate functions for STIM1 and STIM2 in macrophage activation and in the context of inflammationS1P Provokes Tumor Lymphangiogenesis via Macrophage-Derived Mediators Such as IL-1β or Lipocalin-2.Basophils inhibit proliferation of CD4⁺ T cells in autologous and allogeneic mixed lymphocyte reactions and limit disease activity in a murine model of graft versus host disease.Phosphoinositide 3-kinases gamma and delta, linkers of coordinate C5a receptor-Fcgamma receptor activation and immune complex-induced inflammation.The Pyrazole Derivative BTP2 Attenuates IgG Immune Complex-induced Inflammation.C5aR activation in the absence of C5a: A new disease mechanism of autoimmune hemolytic anemia in mice.S1PR1 on tumor-associated macrophages promotes lymphangiogenesis and metastasis via NLRP3/IL-1β.Basophils control T-cell responses and limit disease activity in experimental murine colitis.Killing Is Not Enough: How Apoptosis Hijacks Tumor-Associated Macrophages to Promote Cancer Progression.Targeted inhibition of serotonin type 7 (5-HT7) receptor function modulates immune responses and reduces the severity of intestinal inflammation.IL-6 augments IL-4-induced polarization of primary human macrophages through synergy of STAT3, STAT6 and BATF transcription factorsBoth FcγRIV and FcγRIII are essential receptors mediating type II and type III autoimmune responsesviaFcRγ-LAT-dependent generation of C5aTolerizing CTL by Sustained Hepatic PD-L1 Expression Provides a New Therapy Approach in Mouse SepsisApoptotic tumor cell-derived microRNA-375 uses CD36 to alter the tumor-associated macrophage phenotypeDefective macrophage migration in Gαi2- but not Gαi3-deficient miceMicroRNA-A Tumor Trojan Horse for Tumor-Associated MacrophagesMicroRNAs as Emerging Regulators of Signaling in the Tumor MicroenvironmentMacrophage S1PR1 Signaling Alters Angiogenesis and Lymphangiogenesis During Skin Inflammation
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description
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հետազոտող
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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Shahzad N Syed
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P106
P2798
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P496
0000-0002-6660-6561