Pressure- and volume-induced left ventricular hypertrophies are associated with distinct myocyte phenotypes and differential induction of peptide growth factor mRNAs.
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Alterations in cardiac adrenergic signaling and calcium cycling differentially affect the progression of cardiomyopathyFibroblast growth factor-2 mediates pressure-induced hypertrophic responseProgression from hypertrophic to dilated cardiomyopathy in mice that express a mutant myosin transgene.Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.Evolution and pathophysiology of chronic systolic heart failure.Aortocaval fistula in rat: a unique model of volume-overload congestive heart failure and cardiac hypertrophy.Buckwheat Rutin Inhibits AngII-induced Cardiomyocyte Hypertrophy via Blockade of CaN-dependent Signal Pathway.Elevated mean arterial pressure in the ovariectomized rat was normalized by ET(A) receptor antagonist therapy: absence of cardiac hypertrophy and fibrosis.Fatty acids identified in the Burmese python promote beneficial cardiac growth.Pulmonary artery banding alters the expression of Ca2+ transport proteins in the right atrium in rabbits.Hypertension and heart failure: a dysfunction of systole, diastole or both?Heart failure: when form fails to follow function.Conditional knockout of activin like kinase-1 (ALK-1) leads to heart failure without maladaptive remodeling.Nitric oxide, atrial natriuretic peptide, and cyclic GMP inhibit the growth-promoting effects of norepinephrine in cardiac myocytes and fibroblasts.Prevalence of asymptomatic heart failure in formerly pre-eclamptic women: a cohort study.Myocardial stress and hypertrophy: a complex interface between biophysics and cardiac remodeling.New insights into the molecular phenotype of eccentric hypertrophy.Differential expression of cytokines in the rat heart in response to sustained volume overload.TGF-beta(1) and prepro-ANP mRNAs are differentially regulated in exercise-induced cardiac hypertrophy.Defective intracellular Ca2+ homeostasis contributes to myocyte dysfunction during ventricular remodelling induced by chronic volume overload in rats.Growth patterns of the heart and kidney suggest inter-organ collaboration in facultative fetal growth.Multiple signal transduction pathways link Na+/K+-ATPase to growth-related genes in cardiac myocytes. The roles of Ras and mitogen-activated protein kinases.MK5 haplodeficiency attenuates hypertrophy and preserves diastolic function during remodeling induced by chronic pressure overload in the mouse heart.Candidate mechanical stimuli for hypertrophy during volume overload.Early sequence of cardiac adaptations and growth factor formation in pressure- and volume-overload hypertrophy.Calcineurin inhibition normalizes beta-adrenergic responsiveness in the spontaneously hypertensive rat.Magnetic resonance imaging of progressive cardiomyopathic changes in the db/db mouse.Left ventricular myofilament dysfunction in rat experimental hypertrophy and congestive heart failureEffects of renovascular hypertension on myocardial protein patterns: Analysis by computer-assisted two-dimensional gel electrophoresis
P2860
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P2860
Pressure- and volume-induced left ventricular hypertrophies are associated with distinct myocyte phenotypes and differential induction of peptide growth factor mRNAs.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Pressure- and volume-induced l ...... f peptide growth factor mRNAs.
@en
type
label
Pressure- and volume-induced l ...... f peptide growth factor mRNAs.
@en
prefLabel
Pressure- and volume-induced l ...... f peptide growth factor mRNAs.
@en
P2093
P356
P1433
P1476
Pressure- and volume-induced l ...... f peptide growth factor mRNAs.
@en
P2093
Calderone A
Colucci WS
Izzo NJ Jr
Takahashi N
P304
P356
10.1161/01.CIR.92.9.2385
P407
P577
1995-11-01T00:00:00Z