Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

about

Cordyceps militaris fruit body extract ameliorates membranous glomerulonephritis by attenuating oxidative stress and renal inflammation via the NF-κB pathway.

P2860

Q39893740-E2178A33-AE59-43F9-B76F-3997FF2C777E

P2860

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

Item

http://www.wikidata.org/entity/Q41690085

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

@en

type

Item

label

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

@en

prefLabel

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

@en

P1476

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

@en

P2093

Wen Huang

http://www.w3.org/2001/XMLSchema#string

Wenjing Luo

http://www.w3.org/2001/XMLSchema#string

Xianghong Wu

http://www.w3.org/2001/XMLSchema#string

Xuean Mo

http://www.w3.org/2001/XMLSchema#string

Yanlan Chen

http://www.w3.org/2001/XMLSchema#string

P2860

HIV-1 Tat regulates endothelial cell cycle progression via activation of the Ras/ERK MAPK signaling pathway

Regional gene expression of LOX-1, VCAM-1, and ICAM-1 in aorta of HIV-1 transgenic rats.

Inhibition of telomerase activity alters tight junction protein expression and induces transendothelial migration of HIV-1-infected cells.

Brain inflammation is a common feature of HIV-infected patients without HIV encephalitis or productive brain infection.

HIV-associated neurocognitive disorders persist in the era of potent antiretroviral therapy: CHARTER Study

HIV Tat induces expression of ICAM-1 in HUVECs: implications for miR-221/-222 in HIV-associated cardiomyopathy.

PPARgamma agonist rosiglitazone is neuroprotective after traumatic brain injury via anti-inflammatory and anti-oxidative mechanisms.

Inhibition of oxidative stress-elicited AKT activation facilitates PPARγ agonist-mediated inhibition of stem cell character and tumor growth of liver cancer cells

PPARα and PPARγ protect against HIV-1-induced MMP-9 overexpression via caveolae-associated ERK and Akt signaling.

Blood-brain barrier tight junction disruption in human immunodeficiency virus-1 encephalitis.

P2888

s11010-015-2467-2

P304

173-179

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S11010-015-2467-2

http://www.w3.org/2001/XMLSchema#string

P433

1-2

http://www.w3.org/2001/XMLSchema#string

P478

407

http://www.w3.org/2001/XMLSchema#string

P577

2015-06-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

26048716

http://www.w3.org/2001/XMLSchema#string

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

about

P2860

P2860

Rosiglitazone suppresses HIV-1 Tat-induced vascular inflammation via Akt signaling.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P577

P698

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P577

P698