Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury. - Wikidata - wikimedia - TriplyDB

Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

http://www.wikidata.org/entity/Q41694565

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Distinct Patterns of Wnt3a and Wnt5a Signaling Pathway in the Lung from Rats with Endotoxic Shock Wnt5a attenuates hypoxia-induced pulmonary arteriolar remodeling and right ventricular hypertrophy in mice.Noncanonical WNT-5A signaling impairs endogenous lung repair in COPD LPS-induced inflammatory response is suppressed by Wnt inhibitors, Dickkopf-1 and LGK974 The Wnt Blows: On the Functional Role of Wnt Signaling in Mycobacterium tuberculosis Infection and Beyond Tryptase is involved in the development of early ventilator-induced pulmonary fibrosis in sepsis-induced lung injury.Mitigation of sepsis-induced inflammatory responses and organ injury through targeting Wnt/β-catenin signaling.High attenuation areas on chest computed tomography in community-dwelling adults: the MESA study.Basic fibroblast growth factor activates β-catenin/RhoA signaling in pulmonary fibroblasts with chronic obstructive pulmonary disease in rats.Protective effect of sesquiterpene lactone parthenolide on LPS-induced acute lung injury.Evidence that bone marrow-derived mesenchymal stem cells reduce epithelial permeability following phosgene-induced acute lung injury via activation of wnt3a protein-induced canonical wnt/β-catenin signaling.

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Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

http://www.wikidata.org/entity/Q41694565

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

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type

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Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

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Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

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Early activation of pro-fibrotic WNT5A in sepsis-induced acute lung injury.

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Angela Ramos-Nuez

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Carlos Flores

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Francisco Valladares

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Jesús Villar

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Josefina López-Aguilar

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Lluís Blanch

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Nuria E Cabrera-Benítez

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Sonia García-Hernández

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P2860

The Wingless homolog WNT5A and its receptor Frizzled-5 regulate inflammatory responses of human mononuclear cells induced by microbial stimulation

WNT5A is a regulator of fibroblast proliferation and resistance to apoptosis

WNT1-inducible signaling protein-1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosis

Idiopathic pulmonary fibrosis: an epithelial/fibroblastic cross-talk disorder

Wnt signalling in lung development and diseases

Mechanical ventilation-associated lung fibrosis in acute respiratory distress syndrome: a significant contributor to poor outcome

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

Implication of Toll-like receptor and tumor necrosis factor alpha signaling in septic shock

Wnt signals are transmitted through N-terminally dephosphorylated beta-catenin

Anti-inflammatory activity of a novel family of aryl ureas compounds in an endotoxin-induced airway epithelial cell injury model

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