Il-1β and prostaglandin E2 attenuate the hypercapnic as well as the hypoxic respiratory response via prostaglandin E receptor type 3 in neonatal mice.
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Derivation of Candidate Clinical Decision Rules to Identify Infants at Risk for Central Apnea.CO2-evoked release of PGE2 modulates sighs and inspiration as demonstrated in brainstem organotypic culture.Astrocytes release prostaglandin E2 to modify respiratory network activity.Eupnea, tachypnea, and autoresuscitation in a closed-loop respiratory control model.The Consequences of Preterm Birth and Chorioamnionitis on Brainstem Respiratory Centers: Implications for Neurochemical Development and Altered Functions by Inflammation and Prostaglandins.
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Il-1β and prostaglandin E2 attenuate the hypercapnic as well as the hypoxic respiratory response via prostaglandin E receptor type 3 in neonatal mice.
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Il-1β and prostaglandin E2 att ...... eptor type 3 in neonatal mice.
@en
type
label
Il-1β and prostaglandin E2 att ...... eptor type 3 in neonatal mice.
@en
prefLabel
Il-1β and prostaglandin E2 att ...... eptor type 3 in neonatal mice.
@en
P2860
P1476
Il-1β and prostaglandin E2 att ...... ceptor type 3 in neonatal mice
@en
P2093
Veronica Siljehav
Yuri Shvarev
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00542.2014
P407
P577
2014-09-11T00:00:00Z