BET N-terminal bromodomain inhibition selectively blocks Th17 cell differentiation and ameliorates colitis in mice.
about
Fragment-Based Drug Discovery in the Bromodomain and Extra-Terminal Domain Family.Chemical modulators for epigenome reader domains as emerging epigenetic therapies for cancer and inflammation.Molecular structures guide the engineering of chromatin.BET proteins in abnormal metabolism, inflammation, and the breast cancer microenvironment.Optimization of a "bump-and-hole" approach to allele-selective BET bromodomain inhibition.Immunotherapy With Human Gamma Delta T Cells-Synergistic Potential of Epigenetic Drugs?The Emerging Spectrum of Early Life Exposure-Related Inflammation and Epigenetic Therapy
P2860
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P2860
BET N-terminal bromodomain inhibition selectively blocks Th17 cell differentiation and ameliorates colitis in mice.
description
2017 nî lūn-bûn
@nan
2017年の論文
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2017年論文
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2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
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2017年論文
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2017年论文
@wuu
2017年论文
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2017年论文
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name
BET N-terminal bromodomain inh ...... d ameliorates colitis in mice.
@en
type
label
BET N-terminal bromodomain inh ...... d ameliorates colitis in mice.
@en
prefLabel
BET N-terminal bromodomain inh ...... d ameliorates colitis in mice.
@en
P2093
P2860
P356
P1476
BET N-terminal bromodomain inh ...... d ameliorates colitis in mice.
@en
P2093
Adam Vincek
Alexander N Plotnikov
Anbalagan Jaganathan
David Y Zhang
Elena Rusinova
Feihong Xu
Hongfa Zhu
Huabao Xiong
P2860
P304
P356
10.1073/PNAS.1615601114
P407
P577
2017-03-06T00:00:00Z