Potentiation of reactive oxygen species is a marker for synergistic cytotoxicity of MS-275 and 5-azacytidine in leukemic cells.
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Histone acetylation: novel target for the treatment of acute lymphoblastic leukemiaEpigenomics of leukemia: from mechanisms to therapeutic applicationsOxidative stress induces monocyte necrosis with enrichment of cell-bound albumin and overexpression of endoplasmic reticulum and mitochondrial chaperonesOxidative stress by targeted agents promotes cytotoxicity in hematologic malignancies.Targeting Histone Deacetylases in Diseases: Where Are We?Acetazolamide potentiates the anti-tumor potential of HDACi, MS-275, in neuroblastomaRedox control of leukemia: from molecular mechanisms to therapeutic opportunities.Azacytidine induces necrosis of multiple myeloma cells through oxidative stress.Epigenetic treatment of myelodysplastic syndromes and acute myeloid leukemias.New agents for AML and MDS.Oxidative stress induced in nurses by exposure to preparation and handling of antineoplastic drugs in Mexican hospitals: a multicentric study.Epigenetic regulation in myelodysplastic syndromes: implications for therapy.Therapeutic strategies to enhance the anticancer efficacy of histone deacetylase inhibitors.Entinostat for treatment of solid tumors and hematologic malignancies.Oxidative stress, a common molecular pathway for kidney disease: role of the redox enzyme p66Shc.Generation of reactive oxygen species during apoptosis induced by DNA-damaging agents and/or histone deacetylase inhibitors.Histone Deacetylase Inhibitors as Anticancer Drugs.HDAC inhibitors, MS-275 and salermide, potentiates the anticancer effect of EF24 in human pancreatic cancer cells.Response of esophageal cancer cells to epigenetic inhibitors is mediated via altered thioredoxin activity.Selective inhibition of esophageal cancer cells by combination of HDAC inhibitors and AzacytidineEffects of 5'-azacytidine and alendronate on a hepatocellular carcinoma cell line: a proteomics perspective.Decitabine induces delayed reactive oxygen species (ROS) accumulation in leukemia cells and induces the expression of ROS generating enzymes.Zebularine inhibits the growth of A549 lung cancer cells via cell cycle arrest and apoptosis.In vitro assessment of cytochrome P450 inhibition and induction potential of azacitidine.Epigenetic modification involved in benzene-induced apoptosis through regulating apoptosis-related genes expression.Zebularine-induced apoptosis in Calu-6 lung cancer cells is influenced by ROS and GSH level changes.Screening drug effects in patient-derived cancer cells links organoid responses to genome alterations.Combinatorial effects of histone deacetylase inhibitors (HDACi), vorinostat and entinostat, and adaphostin are characterized by distinct redox alterations.Zebularine inhibits the growth of HeLa cervical cancer cells via cell cycle arrest and caspase-dependent apoptosis.Effects of 5-Azacytidine on Growth and Hypocrellin Production of
P2860
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P2860
Potentiation of reactive oxygen species is a marker for synergistic cytotoxicity of MS-275 and 5-azacytidine in leukemic cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Potentiation of reactive oxyge ...... azacytidine in leukemic cells.
@en
type
label
Potentiation of reactive oxyge ...... azacytidine in leukemic cells.
@en
prefLabel
Potentiation of reactive oxyge ...... azacytidine in leukemic cells.
@en
P2093
P2860
P1433
P1476
Potentiation of reactive oxyge ...... azacytidine in leukemic cells.
@en
P2093
Aaron Mobley
Claudia Miller
Jessica Boklan
Joya Chandra
P2860
P304
P356
10.1016/J.LEUKRES.2007.09.007
P577
2007-11-26T00:00:00Z