Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia. - Wikidata - wikimedia - TriplyDB

Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

http://www.wikidata.org/entity/Q41816001

about

Cancer of the Pancreas: Molecular Pathways and Current Advancement in Treatment Deciphering the role of hedgehog signaling in pancreatic cancer Pancreatic cancer from bench to bedside: molecular pathways and treatment options Current concepts in the diagnosis and pathobiology of intraepithelial neoplasia: A review by organ system KRAS Mutant Pancreatic Cancer: No Lone Path to an Effective Treatment Genetics and biology of pancreatic ductal adenocarcinoma Noncoding RNAs and pancreatic cancer Viro-immune therapy: A new strategy for treatment of pancreatic cancer Developmental Pathways Direct Pancreatic Cancer Initiation from Its Cellular Origin Recent advances in the molecular diagnostics of gastric cancer Perspectives in the treatment of pancreatic adenocarcinoma Pancreatic adenocarcinoma pathology: changing "landscape"Evolution and dynamics of pancreatic cancer progression Inherited pancreatic cancer syndromes Genetic basis of pancreas cancer development and progression: insights from whole-exome and whole-genome sequencing Immune checkpoint and inflammation as therapeutic targets in pancreatic carcinoma Pancreatic Cancer, A Mis-interpreter of the Epigenetic Language Nonviral oncogenic antigens and the inflammatory signals driving early cancer development as targets for cancer immunoprevention A Revised Classification System and Recommendations From the Baltimore Consensus Meeting for Neoplastic Precursor Lesions in the Pancreas The Triple-Code Model for Pancreatic Cancer: Cross Talk Among Genetics, Epigenetics, and Nuclear Structure Anti-pancreatic cancer deliverables from sea: first-hand evidence on the efficacy, molecular targets and mode of action for multifarious polyphenols from five different brown-algae Interleukin-6 is required for pancreatic cancer progression by promoting MAPK signaling activation and oxidative stress resistance Identification of KCa3.1 Channel as a Novel Regulator of Oxidative Phosphorylation in a Subset of Pancreatic Carcinoma Cell Lines Novel Diagnostic and Predictive Biomarkers in Pancreatic Adenocarcinoma.Phase I/II Study of Refametinib (BAY 86-9766) in Combination with Gemcitabine in Advanced Pancreatic cancer.MicroRNA array analysis finds elevated serum miR-1290 accurately distinguishes patients with low-stage pancreatic cancer from healthy and disease controls.Reconstituting development of pancreatic intraepithelial neoplasia from primary human pancreas duct cells.Deep sequencing of cancer-related genes revealed GNAS mutations to be associated with intraductal papillary mucinous neoplasms and its main pancreatic duct dilation.Pathology and molecular genetics of pancreatic neoplasms.Establishment and characterization of 6 novel patient-derived primary pancreatic ductal adenocarcinoma cell lines from Korean pancreatic cancer patients.Genome-scale analysis identifies GJB2 and ERO1LB as prognosis markers in patients with pancreatic cancer.Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.Diversity of Precursor Lesions For Pancreatic Cancer: The Genetics and Biology of Intraductal Papillary Mucinous Neoplasm MAPK signaling is required for dedifferentiation of acinar cells and development of pancreatic intraepithelial neoplasia in mice.Genomic Variations in Pancreatic Cancer and Potential Opportunities for Development of New Approaches for Diagnosis and Treatment.The early detection of pancreatic cancer: what will it take to diagnose and treat curable pancreatic neoplasia?Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions.Oncogenic KRAS signalling in pancreatic cancer.CDK4/6 inhibitors have potent activity in combination with pathway selective therapeutic agents in models of pancreatic cancer.The transcription factor GLI1 modulates the inflammatory response during pancreatic tissue remodeling

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P2860

Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

http://www.wikidata.org/entity/Q41816001

description

2012 nî lūn-bûn

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2012年论文

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2012年论文

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2012年论文

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name

Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

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Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

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prefLabel

Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

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Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.

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Hanno Matthaei

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Jian Wu

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Jun Yu

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Michael Borges

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Mitsuro Kanda

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P2860

Core signaling pathways in human pancreatic cancers revealed by global genomic analyses

Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse

Genome-wide analysis of promoter methylation associated with gene expression profile in pancreatic adenocarcinoma.

Progression model for pancreatic cancer.

Recurrent GNAS mutations define an unexpected pathway for pancreatic cyst development.

When cells get stressed: an integrative view of cellular senescence

Telomere shortening is nearly universal in pancreatic intraepithelial neoplasia

Oncogenic KRas suppresses inflammation-associated senescence of pancreatic ductal cells

Pancreatic duct glands are distinct ductal compartments that react to chronic injury and mediate Shh-induced metaplasia.

Senescence-messaging secretome: SMS-ing cellular stress.

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