Timing in the absence of supraspinal input I: variable, but not fixed, spaced stimulation of the sciatic nerve undermines spinally-mediated instrumental learning.
about
Metaplasticity and behavior: how training and inflammation affect plastic potential within the spinal cord and recovery after injuryLearning from the spinal cord: how the study of spinal cord plasticity informs our view of learningSpinal Plasticity and Behavior: BDNF-Induced Neuromodulation in Uninjured and Injured Spinal CordBrain-derived neurotrophic factor promotes adaptive plasticity within the spinal cord and mediates the beneficial effects of controllable stimulationTiming in the absence of supraspinal input II: regularly spaced stimulation induces a lasting alteration in spinal function that depends on the NMDA receptor, BDNF release, and protein synthesisPeripheral noxious stimulation reduces withdrawal threshold to mechanical stimuli after spinal cord injury: role of tumor necrosis factor alpha and apoptosis.Learning about time within the spinal cord: evidence that spinal neurons can abstract and store an index of regularity.Impact of behavioral control on the processing of nociceptive stimulation.Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injuryMaladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury.Learning about Time within the Spinal Cord II: Evidence that Temporal Regularity Is Encoded by a Spinal Oscillator.Inactivity-induced phrenic and hypoglossal motor facilitation are differentially expressed following intermittent vs. sustained neural apnea.What Is Being Trained? How Divergent Forms of Plasticity Compete To Shape Locomotor Recovery after Spinal Cord Injury.Temporal regularity determines the impact of electrical stimulation on tactile reactivity and response to capsaicin in spinally transected rats.AMPA receptor mediated behavioral plasticity in the isolated rat spinal cord.Timing in the absence of supraspinal input III: regularly spaced cutaneous stimulation prevents and reverses the spinal learning deficit produced by peripheral inflammation.Intermittent apnea elicits inactivity-induced phrenic motor facilitation via a retinoic acid- and protein synthesis-dependent pathway.When Pain Hurts: Nociceptive Stimulation Induces a State of Maladaptive Plasticity and Impairs Recovery after Spinal Cord Injury.Metaplasticity within the spinal cord: Evidence brain-derived neurotrophic factor (BDNF), tumor necrosis factor (TNF), and alterations in GABA function (ionic plasticity) modulate pain and the capacity to learn.Pain Input After Spinal Cord Injury (SCI) Undermines Long-Term Recovery and Engages Signal Pathways That Promote Cell Death.
P2860
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P2860
Timing in the absence of supraspinal input I: variable, but not fixed, spaced stimulation of the sciatic nerve undermines spinally-mediated instrumental learning.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Timing in the absence of supra ...... ediated instrumental learning.
@en
type
label
Timing in the absence of supra ...... ediated instrumental learning.
@en
prefLabel
Timing in the absence of supra ...... ediated instrumental learning.
@en
P2093
P2860
P1433
P1476
Timing in the absence of supra ...... mediated instrumental learning
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2008.07.003
P407
P577
2008-07-04T00:00:00Z