Occurrence of hypermutable Pseudomonas aeruginosa in cystic fibrosis patients is associated with the oxidative stress caused by chronic lung inflammation.
about
Bacterial evolution in PCD and CF patients follows the same mutational stepsEvolution of mutation rates in bacteriaPseudomonas aeruginosa biofilms in cystic fibrosisnfxB as a novel target for analysis of mutation spectra in Pseudomonas aeruginosaGenome analysis of a transmissible lineage of pseudomonas aeruginosa reveals pathoadaptive mutations and distinct evolutionary paths of hypermutatorsEpoxide-mediated CifR repression of cif gene expression utilizes two binding sites in Pseudomonas aeruginosa.Genotypic and phenotypic analyses of a Pseudomonas aeruginosa chronic bronchiectasis isolate reveal differences from cystic fibrosis and laboratory strains.Dynamics of mutator and antibiotic-resistant populations in a pharmacokinetic/pharmacodynamic model of Pseudomonas aeruginosa biofilm treatment.Mutator genes giving rise to decreased antibiotic susceptibility in Pseudomonas aeruginosa.Genomewide identification of genetic determinants of antimicrobial drug resistance in Pseudomonas aeruginosaRole of mutation in Pseudomonas aeruginosa biofilm development.Pseudomonas aeruginosa biofilm infections in cystic fibrosis: insights into pathogenic processes and treatment strategies.Quorum sensing and virulence of Pseudomonas aeruginosa during lung infection of cystic fibrosis patientsEpistatic roles for Pseudomonas aeruginosa MutS and DinB (DNA Pol IV) in coping with reactive oxygen species-induced DNA damage.Evolution of the Pseudomonas aeruginosa mutational resistome in an international Cystic Fibrosis cloneOverexpression of CupB5 activates alginate overproduction in Pseudomonas aeruginosa by a novel AlgW-dependent mechanism.Efflux pumps as antimicrobial resistance mechanisms.Evolution and adaptation in Pseudomonas aeruginosa biofilms driven by mismatch repair system-deficient mutators.Mucoidy, quorum sensing, mismatch repair and antibiotic resistance in Pseudomonas aeruginosa from cystic fibrosis chronic airways infections.Metabolomic profiling reveals biochemical pathways and biomarkers associated with pathogenesis in cystic fibrosis cells.Polymicrobial challenges to Koch's postulates: ecological lessons from the bacterial vaginosis and cystic fibrosis microbiomes.Clinical significance of microbial infection and adaptation in cystic fibrosis.Efficacy and potential for resistance selection of antipseudomonal treatments in a mouse model of lung infection by hypermutable Pseudomonas aeruginosaSocial cheating in Pseudomonas aeruginosa quorum sensing.Evolutionary dynamics of bacteria in a human host environment.Clonal dissemination, emergence of mutator lineages and antibiotic resistance evolution in Pseudomonas aeruginosa cystic fibrosis chronic lung infection.Breaking the rules: bacteria that use several DNA polymerase IIIs.Proposal of a quantitative PCR-based protocol for an optimal Pseudomonas aeruginosa detection in patients with cystic fibrosis.Swimming motility in a longitudinal collection of clinical isolates of Burkholderia cepacia complex bacteria from people with cystic fibrosis.Coexistence and within-host evolution of diversified lineages of hypermutable Pseudomonas aeruginosa in long-term cystic fibrosis infections.Synthesis and structure-activity relationships of novel cationic lipids with anti-inflammatory and antimicrobial activities.The SmeYZ efflux pump of Stenotrophomonas maltophilia contributes to drug resistance, virulence-related characteristics, and virulence in mice.Within-host microevolution of Pseudomonas aeruginosa in Italian cystic fibrosis patients.Influence of high mutation rates on the mechanisms and dynamics of in vitro and in vivo resistance development to single or combined antipseudomonal agents.The properties of spontaneous mutations in the opportunistic pathogen Pseudomonas aeruginosaPseudomonas aeruginosa AmpR: an acute-chronic switch regulator.Phosphorothioate DNA as an antioxidant in bacteria.Anionic fluoroquinolones as antibacterials against biofilm-producing Pseudomonas aeruginosaHypermutable Pseudomonas aeruginosa in Cystic fibrosis patients from two Brazilian cities.Association between hypermutator phenotype, clinical variables, mucoid phenotype, and antimicrobial resistance in Pseudomonas aeruginosa
P2860
Q27334684-3E15C6BE-8F33-4B9C-8582-9DDEDD2BF3CAQ28238561-127EEA8F-1738-44CE-AAB5-B57671465C03Q28299966-2ECF37BF-C9EB-4ACD-BA86-1A2DA911F1CFQ28533745-EB86F507-2E43-4A9F-A009-13084D568779Q28536866-7CB2393C-38CE-4F34-9179-E4A8EF83EA3BQ29346762-DD8656DD-1FCD-4641-8C4A-AD59F07C1A29Q30278439-CC1155A4-D1EF-4C02-A695-6FEA134E053FQ31029779-5CE21E89-A263-40A9-B452-C8A3C039AB47Q33355731-C22884F9-D099-4263-B31B-8CC18B8F4739Q33424246-890CD353-EE3F-49CA-9F6B-4061E1AEDB6AQ33482815-727B7E7C-1EF4-4C72-8C47-87D939AE249DQ33522442-F16FE7D3-1A2E-4808-9DAC-B7A9097375F2Q33559774-7FC649F6-3A76-4D18-8975-0FF8506BAFEDQ33886303-C698C1D6-8557-4543-AECC-180207775D4FQ33911762-13AD45E0-44A2-4F50-9C56-0FDCEE2161EEQ33977944-6CF1F091-C6AF-4AB0-8DBE-5D8084BD5855Q34003782-080EFB49-D002-4FD1-8AAB-AC538CEE1481Q34082906-AB0AED9D-5744-45D8-B072-859582537E32Q34117477-307535AE-B3FD-49E5-8486-3338686AC6EFQ34155281-525C550E-1B43-48CE-9F5A-241086867E19Q34179233-8AB7F35C-571D-47B9-AD89-7F848420AA91Q34491552-49B94BDE-2CBC-4853-9C64-ABC9048FCC53Q34509793-5000C463-3D62-4078-AA7E-3C09BCA06A9DQ34693898-57923EA9-7C5D-4246-BABB-DFB38D8CB47BQ34937408-375F9685-BEF0-448F-A829-4AC2B4E8BA7EQ34948367-05BB9AEB-080F-4811-BD68-1676DDF2A12AQ34953383-D75B7235-2E06-4673-9953-8AA7DBA55483Q35006748-0B4299A0-EA9E-4894-A4C4-4363E8B486C2Q35244339-074CED2C-7CA7-41C6-9920-2570D8317FF5Q35350270-DFF4F7FF-F73E-4B1B-B2A8-A771AA8EA632Q35695892-6885DB09-92FD-4D7C-A541-8BDE42596908Q35746213-CFD3F6B5-79E8-475A-9214-3E30F3F1EB4CQ35813781-0FB12B90-28D8-4B9B-886C-6CA419BD1266Q35879111-F4EBB16D-1A9E-4832-958D-E7DCDF163744Q35885995-21042EDF-2690-4667-B108-D427B7E189DCQ35975439-2D23E78C-67B6-4C21-8AD1-852DEA264F06Q36305614-EF5F27FD-DB13-4FDA-BBDD-2FDD5C7ED2F0Q36580765-9342809F-9C3D-49C2-9D4D-28E2554C2769Q36667671-AC7A4197-3A56-430E-BE08-02E58BCE0418Q36933558-D77A78D8-D08E-4A6B-9FFE-E8183C35FC04
P2860
Occurrence of hypermutable Pseudomonas aeruginosa in cystic fibrosis patients is associated with the oxidative stress caused by chronic lung inflammation.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Occurrence of hypermutable Pse ...... by chronic lung inflammation.
@en
type
label
Occurrence of hypermutable Pse ...... by chronic lung inflammation.
@en
prefLabel
Occurrence of hypermutable Pse ...... by chronic lung inflammation.
@en
P2860
P50
P921
P1476
Occurrence of hypermutable Pse ...... by chronic lung inflammation.
@en
P2093
Bente Riis
Tacjana Pressler
P2860
P304
P356
10.1128/AAC.49.6.2276-2282.2005
P407
P577
2005-06-01T00:00:00Z