NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis. - Wikidata - wikimedia - TriplyDB

NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

http://www.wikidata.org/entity/Q41827067

about

Calcineurin/NFAT pathway mediates wear particle-induced TNF-α release and osteoclastogenesis from mice bone marrow macrophages in vitro.The relative timing of exposure to phagocytosable particulates and to osteoclastogenic cytokines is critically important in the determination of myeloid cell fate Blockade of JNK and NFAT pathways attenuates orthopedic particle-stimulated osteoclastogenesis of human osteoclast precursors and murine calvarial osteolysis.Calcineurin/nuclear factor of activated T cells (NFAT) signaling in cobalt-chromium-molybdenum (CoCrMo) particles-induced tumor necrosis factor-α (TNFα) secretion in MLO-Y4 osteocytes.Effects of orthopedic polymer particles on chemotaxis of macrophages and mesenchymal stem cells.Is there a role for NFAT inhibitors in the prevention of bone destruction?Transcription Factor Ets-2 Acts as a Preinduction Repressor of Interleukin-2 (IL-2) Transcription in Naive T Helper Lymphocytes.Pro-osteoclastic in vitro effect of Polyethylene-like nanoparticles: Involvement in the pathogenesis of implant aseptic loosening.Effect of cyclosporine-A on orthodontic tooth movement in rats.

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NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

http://www.wikidata.org/entity/Q41827067

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

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type

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NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

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prefLabel

NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

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Journal of Orthopaedic Research

P1476

NFAT2 is an essential mediator of orthopedic particle-induced osteoclastogenesis.

@en

P2093

Dominica Foglia

http://www.w3.org/2001/XMLSchema#string

Jesse Otero

http://www.w3.org/2001/XMLSchema#string

John C Clohisy

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Wahid Abu-Amer

http://www.w3.org/2001/XMLSchema#string

Yasuhiro Yamanaka

http://www.w3.org/2001/XMLSchema#string

Yousef Abu-Amer

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P2860

NFAT proteins: key regulators of T-cell development and function

Interaction between c-Rel and the mitogen-activated protein kinase kinase kinase 1 signaling cascade in mediating kappaB enhancer activation

Induction and activation of the transcription factor NFATc1 (NFAT2) integrate RANKL signaling in terminal differentiation of osteoclasts

Transcriptional regulation by calcium, calcineurin, and NFAT

Affinity-driven peptide selection of an NFAT inhibitor more selective than cyclosporin A.

Minireview: the OPG/RANKL/RANK system.

Partners in transcription: NFAT and AP-1.

Role of RANKL and RANK in bone loss and arthritis.

Mechanisms of TNF-alpha- and RANKL-mediated osteoclastogenesis and bone resorption in psoriatic arthritis.

Particle disease. A comprehensive theory of periprosthetic osteolysis: a review.

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1577-1584

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scholarly article

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10.1002/JOR.20714

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12

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26

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2008-12-01T00:00:00Z

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51428390

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18655139

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2613652

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