Fatty acid oxidation in cardiac and skeletal muscle mitochondria is unaffected by deletion of CD36. - Wikidata - wikimedia - TriplyDB

Fatty acid oxidation in cardiac and skeletal muscle mitochondria is unaffected by deletion of CD36.

Fatty acid oxidation in cardiac and skeletal muscle mitochondria is unaffected by deletion of CD36.

http://www.wikidata.org/entity/Q41831699

about

Muscle physiology changes induced by every other day feeding and endurance exercise in mice: effects on physical performance Mitochondrial carnitine palmitoyltransferase 1a (CPT1a) is part of an outer membrane fatty acid transfer complex Novel role of FATP1 in mitochondrial fatty acid oxidation in skeletal muscle cells Mitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes.FAT/CD36 is localized in sarcolemma and in vesicle-like structures in subsarcolemma regions but not in mitochondria MnSOD deficiency results in elevated oxidative stress and decreased mitochondrial function but does not lead to muscle atrophy during aging Activation of AMPKα2 Is Not Required for Mitochondrial FAT/CD36 Accumulation during Exercise.Delineating the role of alterations in lipid metabolism to the pathogenesis of inherited skeletal and cardiac muscle disorders: Thematic Review Series: Genetics of Human Lipid Diseases.Conditional knockout of Mn-SOD targeted to type IIB skeletal muscle fibers increases oxidative stress and is sufficient to alter aerobic exercise capacity.Mitochondrial function and dysfunction in exercise and insulin resistance.Novel ETF dehydrogenase mutations in a patient with mild glutaric aciduria type II and complex II-III deficiency in liver and muscle.Exercise- and training-induced upregulation of skeletal muscle fatty acid oxidation are not solely dependent on mitochondrial machinery and biogenesis.Impaired Energy Production Contributes to Development of Failure in Taurine Deficient Heart.Responses of skeletal muscle lipid metabolism in rat gastrocnemius to hypothyroidism and iodothyronine administration: a putative role for FAT/CD36.Alterations in fatty acid metabolism and sirtuin signaling characterize early type-2 diabetic hearts of fructose-fed rats Complex I generated, mitochondrial matrix-directed superoxide is released from the mitochondria through voltage dependent anion channels.Decreased rates of substrate oxidation ex vivo predict the onset of heart failure and contractile dysfunction in rats with pressure overload.The Implication of PGC-1α on Fatty Acid Transport across Plasma and Mitochondrial Membranes in the Insulin Sensitive Tissues.Rosiglitazone increases fatty acid oxidation and fatty acid translocase (FAT/CD36) but not carnitine palmitoyltransferase I in rat muscle mitochondria.

P2860

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P2860

Fatty acid oxidation in cardiac and skeletal muscle mitochondria is unaffected by deletion of CD36.

http://www.wikidata.org/entity/Q41831699

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Fatty acid oxidation in cardia ...... naffected by deletion of CD36.

@en

type

label

Fatty acid oxidation in cardia ...... naffected by deletion of CD36.

@en

prefLabel

Fatty acid oxidation in cardia ...... naffected by deletion of CD36.

@en

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P356

J.ABB.2007.08.020

P1433

Archives of Biochemistry and Biophysics

P1476

Fatty acid oxidation in cardia ...... naffected by deletion of CD36.

@en

P2093

Charles L Hoppel

http://www.w3.org/2001/XMLSchema#string

Janos Kerner

http://www.w3.org/2001/XMLSchema#string

Kristen L King

http://www.w3.org/2001/XMLSchema#string

Maria Febbraio

http://www.w3.org/2001/XMLSchema#string

Mariana Rosca

http://www.w3.org/2001/XMLSchema#string

William C Stanley

http://www.w3.org/2001/XMLSchema#string

P2860

Fatty acid import into mitochondria

A novel type of short- and medium-chain acyl-CoA hydrolases in brown adipose tissue mitochondria

A novel function for fatty acid translocase (FAT)/CD36: involvement in long chain fatty acid transfer into the mitochondria

Diabetes or peroxisome proliferator-activated receptor alpha agonist increases mitochondrial thioesterase I activity in heart

CD36 mediates the cardiovascular action of growth hormone-releasing peptides in the heart

Electrostimulation enhances FAT/CD36-mediated long-chain fatty acid uptake by isolated rat cardiac myocytes.

Preservation of cardiolipin content during aging in rat heart interfibrillar mitochondria.

Fatty acid translocase/CD36 deficiency does not energetically or functionally compromise hearts before or after ischemia.

Identification of fatty acid translocase on human skeletal muscle mitochondrial membranes: essential role in fatty acid oxidation.

Mitochondrial long chain fatty acid oxidation, fatty acid translocase/CD36 content and carnitine palmitoyltransferase I activity in human skeletal muscle during aerobic exercise.

P304

234-238

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P31

scholarly article

P356

10.1016/J.ABB.2007.08.020

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P433

2

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467

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2007-08-29T00:00:00Z

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5940996

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17904092

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2702466

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