Fatty acid oxidation in cardiac and skeletal muscle mitochondria is unaffected by deletion of CD36.
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Muscle physiology changes induced by every other day feeding and endurance exercise in mice: effects on physical performanceMitochondrial carnitine palmitoyltransferase 1a (CPT1a) is part of an outer membrane fatty acid transfer complexNovel role of FATP1 in mitochondrial fatty acid oxidation in skeletal muscle cellsMitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes.FAT/CD36 is localized in sarcolemma and in vesicle-like structures in subsarcolemma regions but not in mitochondriaMnSOD deficiency results in elevated oxidative stress and decreased mitochondrial function but does not lead to muscle atrophy during agingActivation of AMPKα2 Is Not Required for Mitochondrial FAT/CD36 Accumulation during Exercise.Delineating the role of alterations in lipid metabolism to the pathogenesis of inherited skeletal and cardiac muscle disorders: Thematic Review Series: Genetics of Human Lipid Diseases.Conditional knockout of Mn-SOD targeted to type IIB skeletal muscle fibers increases oxidative stress and is sufficient to alter aerobic exercise capacity.Mitochondrial function and dysfunction in exercise and insulin resistance.Novel ETF dehydrogenase mutations in a patient with mild glutaric aciduria type II and complex II-III deficiency in liver and muscle.Exercise- and training-induced upregulation of skeletal muscle fatty acid oxidation are not solely dependent on mitochondrial machinery and biogenesis.Impaired Energy Production Contributes to Development of Failure in Taurine Deficient Heart.Responses of skeletal muscle lipid metabolism in rat gastrocnemius to hypothyroidism and iodothyronine administration: a putative role for FAT/CD36.Alterations in fatty acid metabolism and sirtuin signaling characterize early type-2 diabetic hearts of fructose-fed ratsComplex I generated, mitochondrial matrix-directed superoxide is released from the mitochondria through voltage dependent anion channels.Decreased rates of substrate oxidation ex vivo predict the onset of heart failure and contractile dysfunction in rats with pressure overload.The Implication of PGC-1α on Fatty Acid Transport across Plasma and Mitochondrial Membranes in the Insulin Sensitive Tissues.Rosiglitazone increases fatty acid oxidation and fatty acid translocase (FAT/CD36) but not carnitine palmitoyltransferase I in rat muscle mitochondria.
P2860
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P2860
Fatty acid oxidation in cardiac and skeletal muscle mitochondria is unaffected by deletion of CD36.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Fatty acid oxidation in cardia ...... naffected by deletion of CD36.
@en
type
label
Fatty acid oxidation in cardia ...... naffected by deletion of CD36.
@en
prefLabel
Fatty acid oxidation in cardia ...... naffected by deletion of CD36.
@en
P2093
P2860
P1476
Fatty acid oxidation in cardia ...... naffected by deletion of CD36.
@en
P2093
Charles L Hoppel
Janos Kerner
Kristen L King
Maria Febbraio
Mariana Rosca
William C Stanley
P2860
P304
P356
10.1016/J.ABB.2007.08.020
P407
P577
2007-08-29T00:00:00Z