Evidence of inflammatory cell involvement in brain arteriovenous malformations. - Wikidata - wikimedia - TriplyDB

Evidence of inflammatory cell involvement in brain arteriovenous malformations.

Evidence of inflammatory cell involvement in brain arteriovenous malformations.

http://www.wikidata.org/entity/Q41842014

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Differential gene expression in relation to the clinical characteristics of human brain arteriovenous malformations Biology of cerebral arteriovenous malformations with a focus on inflammation.Case report of a de novo brainstem arteriovenous malformation in an 18-year-old male and review of the literature.Imaging Inflammation in Cerebrovascular Disease Biology of vascular malformations of the brain.Brain arteriovenous malformation modeling, pathogenesis, and novel therapeutic targets.VEGF Induces More Severe Cerebrovascular Dysplasia in Endoglin than in Alk1 Mice.Hemorrhage rates and risk factors in the natural history course of brain arteriovenous malformations.EPHB4 gene polymorphisms and risk of intracranial hemorrhage in patients with brain arteriovenous malformations.Bone marrow-derived cells contribute to vascular endothelial growth factor-induced angiogenesis in the adult mouse brain by supplying matrix metalloproteinase-9 Distinctive distribution of lymphocytes in unruptured and previously untreated brain arteriovenous malformation Novel brain arteriovenous malformation mouse models for type 1 hereditary hemorrhagic telangiectasia.Gene expression profiling of blood in brain arteriovenous malformation patients.Silent intralesional microhemorrhage as a risk factor for brain arteriovenous malformation rupture.The Role of Macrophage in the Pathogenesis of Brain Arteriovenous Malformation.Vascular Integrity in the Pathogenesis of Brain Arteriovenous Malformation.Reduced mural cell coverage and impaired vessel integrity after angiogenic stimulation in the Alk1-deficient brain.Endoglin deficiency in bone marrow is sufficient to cause cerebrovascular dysplasia in the adult mouse after vascular endothelial growth factor stimulation.Safety and technique of ferumoxytol administration for MRI.Histopathological Features of Brain Arteriovenous Malformations in Japanese Patients.Morphometric characterization of brain arteriovenous malformations for clinical and radiological studies to identify silent intralesional microhemorrhages Genetic considerations relevant to intracranial hemorrhage and brain arteriovenous malformations.Nonischemic cerebral venous hypertension promotes a pro-angiogenic stage through HIF-1 downstream genes and leukocyte-derived MMP-9.Immune response in human cerebral cavernous malformations.Endothelial signaling and the molecular basis of arteriovenous malformation.Brain arteriovenous malformation pathogenesis: a response-to-injury paradigm.Single nucleotide polymorphisms associated with sporadic brain arteriovenous malformations: where do we stand?The emerging role of ferumoxytol-enhanced MRI in the management of cerebrovascular lesions.Molecular imaging of cerebrovascular lesions.Emerging applications for ferumoxytol as a contrast agent in MRI.Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin.Persistent infiltration and pro-inflammatory differentiation of monocytes cause unresolved inflammation in brain arteriovenous malformation.Pathogenesis of Brain Arteriovenous Malformations.Brain arteriovenous malformation biology relevant to hemorrhage and implication for therapeutic development.Quantitative in vitro assay to measure neutrophil adhesion to activated primary human microvascular endothelial cells under static conditions.Molecular, Cellular, and Genetic Determinants of Sporadic Brain Arteriovenous Malformations.Ferumoxytol-enhanced MRI to Image Inflammation within Human Brain Arteriovenous Malformations: A Pilot Investigation.Analysis of the Expression of Angioarchitecture-related Factors in Patients with Cerebral Arteriovenous Malformation.Focal neurons: another source of vascular endothelial growth factor in brain arteriovenous malformation tissues?Ruptured brain arteriovenous malformations associated with aneurysms: safety and efficacy of selective embolization in the acute phase of hemorrhage.

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P2860

Evidence of inflammatory cell involvement in brain arteriovenous malformations.

http://www.wikidata.org/entity/Q41842014

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年论文

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2008年论文

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name

Evidence of inflammatory cell involvement in brain arteriovenous malformations.

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type

label

Evidence of inflammatory cell involvement in brain arteriovenous malformations.

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Evidence of inflammatory cell involvement in brain arteriovenous malformations.

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Evidence of inflammatory cell involvement in brain arteriovenous malformations.

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P2093

Andrew W Bollen

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Christopher F Dowd

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Guo-Yuan Yang

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Nicholas M Barbaro

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Tomoki Hashimoto

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Wei Zhu

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William L Young

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Yongmei Chen

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P2860

Gelatinase B functions as regulator and effector in leukocyte biology

Macrophage myeloperoxidase regulation by granulocyte macrophage colony-stimulating factor in human atherosclerosis and implications in acute coronary syndromes

Myeloperoxidase: friend and foe

Arteriovenous malformations.

Myeloperoxidase.

Role of CD8(+) lymphocytes in control of simian immunodeficiency virus infection and resistance to rechallenge after transient early antiretroviral treatment

Myeloperoxidase, a catalyst for lipoprotein oxidation, is expressed in human atherosclerotic lesions

Myeloperoxidase and cardiovascular disease.

Inhibition of plaque neovascularization reduces macrophage accumulation and progression of advanced atherosclerosis

Neutrophil protein kinase Cdelta as a mediator of stroke-reperfusion injury.

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1340-9; discussion 1349-50

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10.1227/01.NEU.0000333306.64683.B5

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6

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62

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Michael T Lawton

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2008-06-01T00:00:00Z

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23287448

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18825001

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arteriovenous malformation

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2582017

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