about
Positive selection acting on splicing motifs reflects compensatory evolutionSplicing factor SFRS1 recognizes a functionally diverse landscape of RNA transcriptsContext-dependent control of alternative splicing by RNA-binding proteinsA machine learning strategy to identify candidate binding sites in human protein-coding sequencePre-mRNA secondary structures influence exon recognition.Accurate splice site prediction using support vector machinesIntronic Alus influence alternative splicing.Functional selection and systematic analysis of intronic splicing elements identify active sequence motifs and associated splicing factors.The intronic splicing code: multiple factors involved in ATM pseudoexon definition.TIA1 prevents skipping of a critical exon associated with spinal muscular atrophy.Splicing of a critical exon of human Survival Motor Neuron is regulated by a unique silencer element located in the last intron.Dual role of G-runs and hnRNP F in the regulation of a mutation-activated pseudoexon in the fibrinogen gamma-chain transcript.Interplay between DMD point mutations and splicing signals in Dystrophinopathy phenotypes.A systematic analysis of intronic sequences downstream of 5' splice sites reveals a widespread role for U-rich motifs and TIA1/TIAL1 proteins in alternative splicing regulation.Evolutionary divergence of exon flanks: a dissection of mutability and selection.Functional single nucleotide polymorphism-based association studies.Defective splicing, disease and therapy: searching for master checkpoints in exon definition.Downstream intronic splicing enhancers.Sequence variation between 462 human individuals fine-tunes functional sites of RNA processingSplice site strength-dependent activity and genetic buffering by poly-G runs.Alternative splicing: role of pseudoexons in human disease and potential therapeutic strategies.Role of pseudoexons and pseudointrons in human cancer.A functional conserved intronic G run in HIV-1 intron 3 is critical to counteract APOBEC3G-mediated host restriction.RNA structure is a key regulatory element in pathological ATM and CFTR pseudoexon inclusion events.Exon inclusion is dependent on predictable exonic splicing enhancersIdentification of motifs that function in the splicing of non-canonical introns.Splicing of designer exons reveals unexpected complexity in pre-mRNA splicingIntronic motif pairs cooperate across exons to promote pre-mRNA splicing.Quantitative evaluation of all hexamers as exonic splicing elementsStoichiometry of a regulatory splicing complex revealed by single-molecule analyses.Ab initio identification of functionally interacting pairs of cis-regulatory elements.TP53 mutations in canine brain tumors.Saturation mutagenesis reveals manifold determinants of exon definition.RNA-Seq Analysis of an Antisense Sequence Optimized for Exon Skipping in Duchenne Patients Reveals No Off-Target Effect.Integrating haplotypes and single genetic variability effects of the Pax7 gene on growth traits in two cattle breeds.Discovering weighted patterns in intron sequences using self-adaptive harmony search and back-propagation algorithms.Splicing and transcription touch base: co-transcriptional spliceosome assembly and function.Splicing and Disease
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Dichotomous splicing signals in exon flanks.
@en
type
label
Dichotomous splicing signals in exon flanks.
@en
prefLabel
Dichotomous splicing signals in exon flanks.
@en
P2093
P2860
P356
P1433
P1476
Dichotomous splicing signals in exon flanks.
@en
P2093
Christina S Leslie
Lawrence A Chasin
Xiang H-F Zhang
P2860
P304
P356
10.1101/GR.3217705
P577
2005-06-01T00:00:00Z