miRNA-21 is dysregulated in response to vein grafting in multiple models and genetic ablation in mice attenuates neointima formation. - Wikidata - wikimedia - TriplyDB

miRNA-21 is dysregulated in response to vein grafting in multiple models and genetic ablation in mice attenuates neointima formation.

miRNA-21 is dysregulated in response to vein grafting in multiple models and genetic ablation in mice attenuates neointima formation.

http://www.wikidata.org/entity/Q41865858

about

Adventitial vessel growth and progenitor cells activation in an ex vivo culture system mimicking human saphenous vein wall strain after coronary artery bypass grafting Resveratrol-Induced Vascular Progenitor Differentiation towards Endothelial Lineage via MiR-21/Akt/β-Catenin Is Protective in Vessel Graft Models High expression of ubiquitin-specific peptidase 39 is associated with the development of vascular remodeling.MiR-551b-5p Contributes to Pathogenesis of Vein Graft Failure via Upregulating Early Growth Response-1 Expression.A selective microRNA-based strategy inhibits restenosis while preserving endothelial function.Role of flow-sensitive microRNAs in endothelial dysfunction and atherosclerosis: mechanosensitive athero-miRs Crosstalk between TGF-β/Smad3 and BMP/BMPR2 signaling pathways via miR-17-92 cluster in carotid artery restenosis Reducing In-Stent Restenosis: Therapeutic Manipulation of miRNA in Vascular Remodeling and Inflammation.Local MicroRNA Modulation Using a Novel Anti-miR-21-Eluting Stent Effectively Prevents Experimental In-Stent Restenosis Local Anti-miR Delivery: The Latest in the Arsenal of Drug-Eluting Stents.Novel potential targets for prevention of arterial restenosis: insights from the pre-clinical research.Nanomedicine Meets microRNA: Current Advances in RNA-Based Nanotherapies for Atherosclerosis.Epigenetics and Vascular Diseases: Influence of Non-coding RNAs and Their Clinical Implications.Macrophage-stimulated microRNA expression in mural cells promotes transplantation-induced neointima formation.Upregulation of miR-126-3p promotes human saphenous vein endothelial cell proliferation in vitro and prevents vein graft neointimal formation ex vivo and in vivo.MicroRNA-33 protects against neointimal hyperplasia induced by arterial mechanical stretch in the grafted vein.Association between hypertension and circulating vascular-related microRNAs.

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P2860

miRNA-21 is dysregulated in response to vein grafting in multiple models and genetic ablation in mice attenuates neointima formation.

http://www.wikidata.org/entity/Q41865858

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miRNA-21 is dysregulated in re ...... ttenuates neointima formation.

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European Heart Journal

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miRNA-21 is dysregulated in re ...... ttenuates neointima formation.

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P2093

Andrew H Baker

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Brian C Cooley

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Crawford A Halliday

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Eva van Rooij

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John D McClure

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Junxi Wu

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Katie M White

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Keith E Robertson

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Robert A McDonald

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Ruifaug Lu

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P2860

MicroRNA-21 blocks abdominal aortic aneurysm development and nicotine-augmented expansion

The knockout of miR-143 and -145 alters smooth muscle cell maintenance and vascular homeostasis in mice: correlates with human disease

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

MicroRNA-145, a novel smooth muscle cell phenotypic marker and modulator, controls vascular neointimal lesion formation

The functions of animal microRNAs

MicroRNA-21 regulates vascular smooth muscle cell function via targeting tropomyosin 1 in arteriosclerosis obliterans of lower extremities

Long-term patency of saphenous vein and left internal mammary artery grafts after coronary artery bypass surgery: results from a Department of Veterans Affairs Cooperative Study

An essential role of PDCD4 in vascular smooth muscle cell apoptosis and proliferation: implications for vascular disease

Inactivation of the tumour suppressor, PTEN, in smooth muscle promotes a pro-inflammatory phenotype and enhances neointima formation

A gene therapy strategy using a transcription factor decoy of the E2F binding site inhibits smooth muscle proliferation in vivo.

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1636-1643

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10.1093/EURHEARTJ/EHT105

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22

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34

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Sheila E. Francis

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2013-03-25T00:00:00Z

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236081715

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23530023

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3675389

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