Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.
about
Aberrant cell cycle reentry in human and experimental inclusion body myositis and polymyositisCalcium signaling and amyloid toxicity in Alzheimer disease.Nonimmune mechanisms of muscle damage in myositis: role of the endoplasmic reticulum stress response and autophagy in the disease pathogenesis.Expression of human amyloid precursor protein in the skeletal muscles of Drosophila results in age- and activity-dependent muscle weakness.Endoplasmic reticulum Ca(2+) handling in excitable cells in health and diseaseAnalyzing and Quantifying the Gain-of-Function Enhancement of IP3 Receptor Gating by Familial Alzheimer's Disease-Causing Mutants in Presenilins.Mitochondrial dysfunction in skeletal muscle of amyloid precursor protein-overexpressing mice.The Phosphorylation Profile of Myosin Binding Protein-C Slow is Dynamically Regulated in Slow-Twitch Muscles in Health and Disease.Amyloid beta peptide 22-35 induces a negative inotropic effect on isolated rat hearts.Pathogenesis, classification and treatment of inflammatory myopathies.Ryanodine receptors: physiological function and deregulation in Alzheimer disease.Phosphoinositides in Ca(2+) signaling and excitation-contraction coupling in skeletal muscle: an old player and newcomers.The Immune Response and the Pathogenesis of Idiopathic Inflammatory Myositis: a Critical Review.Cross-Sectional and Prospective Associations Between β-Amyloid in the Brain and Chair Rise Performance in Nondementia Older Adults With Spontaneous Memory Complaints.Altered Ca2+ homeostasis in the skeletal muscle of DJ-1 null mice.Increased amyloid β-peptide uptake in skeletal muscle is induced by hyposialylation and may account for apoptosis in GNE myopathy.Primary over-expression of AβPP in muscle does not lead to the development of inclusion body myositis in a new lineage of the MCK-AβPP transgenic mouse.Provision of an explanation for the inefficacy of immunotherapy in sporadic inclusion body myositis: quantitative assessment of inflammation and β-amyloid in the muscle.Multiple mechanisms of dimethyl fumarate in amyloid β-induced neurotoxicity in human neuronal cells.A mathematical model for the effects of amyloid beta on intracellular calcium
P2860
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P2860
Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.
@en
type
label
Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.
@en
prefLabel
Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.
@en
P2093
P2860
P1476
Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle
@en
P2093
Alexander Shtifman
Christopher W Ward
Derek R Laver
Frank M LaFerla
Henry W Querfurth
Jose R Lopez
Masashi Kitazawa
Noriaki Ikemoto
P2860
P304
P356
10.1016/J.NEUROBIOLAGING.2008.11.003
P577
2008-12-23T00:00:00Z