The key sigma factor of transition phase, SigH, controls sporulation, metabolism, and virulence factor expression in Clostridium difficile.
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The Regulatory Networks That Control Clostridium difficile Toxin SynthesisArrangement of the Clostridium baratii F7 toxin gene cluster with identification of a σ factor that recognizes the botulinum toxin gene cluster promotersC. difficile 630Δerm Spo0A regulates sporulation, but does not contribute to toxin production, by direct high-affinity binding to target DNAGlobal transcriptional control by glucose and carbon regulator CcpA in Clostridium difficile.Eubacterial SpoVG homologs constitute a new family of site-specific DNA-binding proteins.Transcriptome profiling analysis reveals metabolic changes across various growth phases in Bacillus pumilus BA06Pleiotropic role of the RNA chaperone protein Hfq in the human pathogen Clostridium difficileCharacterization of an insecticidal toxin and pathogenicity of Pseudomonas taiwanensis against insects.Conserved oligopeptide permeases modulate sporulation initiation in Clostridium difficile.Initiation of sporulation in Clostridium difficile: a twist on the classic modelUsing phenotype microarrays to determine culture conditions that induce or repress toxin production by Clostridium difficile and other microorganisms.Genome-wide identification of regulatory RNAs in the human pathogen Clostridium difficile.Global analysis of the sporulation pathway of Clostridium difficile.Genome-wide analysis of cell type-specific gene transcription during spore formation in Clostridium difficile.The spore differentiation pathway in the enteric pathogen Clostridium difficileSpo0A differentially regulates toxin production in evolutionarily diverse strains of Clostridium difficileCharacterization of the SigD regulon of C. difficile and its positive control of toxin production through the regulation of tcdR.Cyclic di-GMP riboswitch-regulated type IV pili contribute to aggregation of Clostridium difficile.Semiquantitative analysis of clinical heat stress in Clostridium difficile strain 630 using a GeLC/MS workflow with emPAI quantitationIntegration of metabolism and virulence in Clostridium difficile.The Clostridium sporulation programs: diversity and preservation of endospore differentiationHigh-throughput analysis of gene essentiality and sporulation in Clostridium difficileClostridium difficile infection: toxins and non-toxin virulence factors, and their contributions to disease establishment and host response.CdtR Regulates TcdA and TcdB Production in Clostridium difficileClostridium difficile colitis: pathogenesis and host defenceA Recombination Directionality Factor Controls the Cell Type-Specific Activation of σK and the Fidelity of Spore Development in Clostridium difficileModulation of toxin production by the flagellar regulon in Clostridium difficile.A genetic switch controls the production of flagella and toxins in Clostridium difficileProline-dependent regulation of Clostridium difficile Stickland metabolism.The Clostridium difficile cpr locus is regulated by a noncontiguous two-component system in response to type A and B lantibiotics.A novel regulator controls Clostridium difficile sporulation, motility and toxin productionCodY-Dependent Regulation of Sporulation in Clostridium difficile.The second messenger cyclic Di-GMP regulates Clostridium difficile toxin production by controlling expression of sigDAdaptive strategies and pathogenesis of Clostridium difficile from in vivo transcriptomicsThe Phosphotransfer Protein CD1492 Represses Sporulation Initiation in Clostridium difficile.Transcriptional analysis of degenerate strain Clostridium beijerinckii DG-8052 reveals a pleiotropic response to CaCO3-associated recovery of solvent production.Biotechnological applications of mobile group II introns and their reverse transcriptases: gene targeting, RNA-seq, and non-coding RNA analysis.Sigma Factor Regulated Cellular Response in a Non-solvent Producing Clostridium beijerinckii Degenerated Strain: A Comparative Transcriptome Analysis.Effect of tcdR Mutation on Sporulation in the Epidemic Clostridium difficile Strain R20291.Variations in virulence and molecular biology among emerging strains of Clostridium difficile.
P2860
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P2860
The key sigma factor of transition phase, SigH, controls sporulation, metabolism, and virulence factor expression in Clostridium difficile.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
The key sigma factor of transi ...... sion in Clostridium difficile.
@en
type
label
The key sigma factor of transi ...... sion in Clostridium difficile.
@en
prefLabel
The key sigma factor of transi ...... sion in Clostridium difficile.
@en
P2093
P2860
P356
P1476
The key sigma factor of transi ...... sion in Clostridium difficile.
@en
P2093
Bruno Dupuy
Isabelle Martin-Verstraete
Laure Saujet
Marc Monot
Olga Soutourina
P2860
P304
P356
10.1128/JB.00272-11
P577
2011-05-13T00:00:00Z