Deletion of intestinal epithelial cell STAT3 promotes T-lymphocyte STAT3 activation and chronic colitis following acute dextran sodium sulfate injury in mice.
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Intestinal epithelium in inflammatory bowel diseaseDendritic cells from Crohn's disease patients show aberrant STAT1 and STAT3 signalingRon receptor signaling is protective against DSS-induced colitis in mice.Prohibitin 1 modulates mitochondrial function of Stat3.Intestinal epithelial expression of TNFAIP3 results in microbial invasion of the inner mucus layer and induces colitis in IL-10-deficient mice.Role of interleukin-22 in inflammatory bowel diseaseNoncanonical STAT3 activation regulates excess TGF-β1 and collagen I expression in muscle of stricturing Crohn's disease.STAT3 polymorphism and Helicobacter pylori CagA strains with higher number of EPIYA-C segments independently increase the risk of gastric cancer.Intestinal Epithelial Cell Tyrosine Kinase 2 Transduces IL-22 Signals To Protect from Acute Colitis.The AIM2 inflammasome is a central regulator of intestinal homeostasis through the IL-18/IL-22/STAT3 pathway.JAK-STAT and intestinal mucosal immunology.Immune deficiency vs. immune excess in inflammatory bowel diseases-STAT3 as a rheo-STAT of intestinal homeostasis.Genomic dissection of conserved transcriptional regulation in intestinal epithelial cells.Though Active on RINm5F Insulinoma Cells and Cultured Pancreatic Islets, Recombinant IL-22 Fails to Modulate Cytotoxicity and Disease in a Protocol of Streptozotocin-Induced Experimental DiabetesTofacitinib for the treatment of ulcerative colitis.STAT3 activation in infection and infection-associated cancer.Classic IL-6R signalling is dispensable for intestinal epithelial proliferation and repair.Epimorphin(-/-) mice are protected, in part, from acute colitis via decreased interleukin 6 signaling.Acceleration of pancreatic tumorigenesis under immunosuppressive microenvironment induced by Reg3g overexpressionMicroscopic colitis-microbiome, barrier function and associated diseases.Glutathione Peroxidase 2, a Selenoprotein Exhibiting a Dual Personality in Preventing and Promoting CancerGPx2 Induction Is Mediated Through STAT Transcription Factors During Acute Colitis
P2860
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P2860
Deletion of intestinal epithelial cell STAT3 promotes T-lymphocyte STAT3 activation and chronic colitis following acute dextran sodium sulfate injury in mice.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Deletion of intestinal epithel ...... sodium sulfate injury in mice.
@en
type
label
Deletion of intestinal epithel ...... sodium sulfate injury in mice.
@en
prefLabel
Deletion of intestinal epithel ...... sodium sulfate injury in mice.
@en
P2093
P2860
P1476
Deletion of intestinal epithel ...... sodium sulfate injury in mice.
@en
P2093
Ingrid Jurickova
Lee A Denson
Margaret Collins
Tara A Willson
P2860
P304
P356
10.1097/MIB.0B013E31828028AD
P577
2013-03-01T00:00:00Z