RIM-binding protein links synaptic homeostasis to the stabilization and replenishment of high release probability vesicles
about
A novel synaptic plasticity rule explains homeostasis of neuromuscular transmissionRab Interacting Molecules 2 and 3 Directly Interact with the Pore-Forming CaV1.3 Ca2+ Channel Subunit and Promote Its Membrane Expression.A single-cross, RNA interference-based genetic tool for examining the long-term maintenance of homeostatic plasticityC-terminal Src Kinase Gates Homeostatic Synaptic Plasticity and Regulates Fasciclin II Expression at the Drosophila Neuromuscular Junction.Regulation of synaptic activity by snapin-mediated endolysosomal transport and sorting.Fife organizes synaptic vesicles and calcium channels for high-probability neurotransmitter releaseReversible Recruitment of a Homeostatic Reserve Pool of Synaptic Vesicles Underlies Rapid Homeostatic Plasticity of Quantal Content.Determining synaptic parameters using high-frequency activationRIM-binding protein 2 regulates release probability by fine-tuning calcium channel localization at murine hippocampal synapses.Synaptic Vesicle Proteins and Active Zone Plasticity.Mechanisms of homeostatic plasticity in the excitatory synapse.Electrophysiological analysis of synaptic transmission in Drosophila.Vertebrate Presynaptic Active Zone Assembly: a Role Accomplished by Diverse Molecular and Cellular Mechanisms.α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.Composition and Control of a Deg/ENaC Channel during Presynaptic Homeostatic Plasticity.Homeostatic synaptic depression is achieved through a regulated decrease in presynaptic calcium channel abundance.MCTP is an ER-resident calcium sensor that stabilizes synaptic transmission and homeostatic plasticityDisparate Postsynaptic Induction Mechanisms Ultimately Converge to Drive the Retrograde Enhancement of Presynaptic Efficacy.RIM-Binding Protein 2 Promotes a Large Number of CaV1.3 Ca2+-Channels and Contributes to Fast Synaptic Vesicle Replenishment at Hair Cell Active Zones.The Maintenance of Synaptic Homeostasis at the Drosophila Neuromuscular Junction Is Reversible and Sensitive to High Temperature.A postsynaptic Pi3K-CII dependent signaling controller for presynaptic homeostatic plasticity.Muscle nicotinic acetylcholine receptors may mediate trans-synaptic signaling at the mouse neuromuscular junction.Efficient stimulus-secretion coupling at ribbon synapses requires RIM-binding protein tethering of L-type Ca2+ channels.Dysbindin links presynaptic proteasome function to homeostatic recruitment of low release probability vesicles.Synapse-specific and compartmentalized expression of presynaptic homeostatic potentiation.RIM-binding proteins recruit BK-channels to presynaptic release sites adjacent to voltage-gated Ca-channels
P2860
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P2860
RIM-binding protein links synaptic homeostasis to the stabilization and replenishment of high release probability vesicles
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
RIM-binding protein links syna ...... h release probability vesicles
@en
type
label
RIM-binding protein links syna ...... h release probability vesicles
@en
prefLabel
RIM-binding protein links syna ...... h release probability vesicles
@en
P2860
P1433
P1476
RIM-binding protein links syna ...... h release probability vesicles
@en
P2093
Graeme W Davis
Martin Müller
P2860
P304
P356
10.1016/J.NEURON.2015.01.024
P407
P50
P577
2015-02-19T00:00:00Z