Infarct size reduction by cyclosporine A at reperfusion involves inhibition of the mitochondrial permeability transition pore but does not improve mitochondrial respiration.
about
Postconditioning signalling in the heart: mechanisms and translatabilityIschemia and reperfusion induce differential expression of calpastatin and its homologue high molecular weight calmodulin-binding protein in murine cardiomyocytes.Cyclosporine A reduces microvascular obstruction and preserves left ventricular function deterioration following myocardial ischemia and reperfusion.Extracellular signalling molecules in the ischaemic/reperfused heart - druggable and translatable for cardioprotection?Pleiotropic, heart rate-independent cardioprotection by ivabradine.Cardiomyocyte mitochondria as targets of humoral factors released by remote ischemic preconditioning.Preserved cardiac mitochondrial function and reduced ischaemia/reperfusion injury afforded by chronic continuous hypoxia: role of opioid receptors.Sevoflurane postconditioning affects post-ischaemic myocardial mitochondrial ATP-sensitive potassium channel function and apoptosis in ageing rats.Protective role of cyclosporine A and minocycline on mitochondrial disequilibrium-related podocyte injury and proteinuria occurrence induced by adriamycin.
P2860
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P2860
Infarct size reduction by cyclosporine A at reperfusion involves inhibition of the mitochondrial permeability transition pore but does not improve mitochondrial respiration.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Infarct size reduction by cycl ...... ove mitochondrial respiration.
@en
type
label
Infarct size reduction by cycl ...... ove mitochondrial respiration.
@en
prefLabel
Infarct size reduction by cycl ...... ove mitochondrial respiration.
@en
P2093
P2860
P356
P1476
Infarct size reduction by cycl ...... ove mitochondrial respiration.
@en
P2093
Andreas Skyschally
Gerd Heusch
Nilguen Gedik
P2860
P304
P356
10.5114/AOMS.2013.38175
P577
2013-12-26T00:00:00Z