Mechanism of liponecrosis, a distinct mode of programmed cell death. - Wikidata - wikimedia - TriplyDB

Mechanism of liponecrosis, a distinct mode of programmed cell death.

Mechanism of liponecrosis, a distinct mode of programmed cell death.

http://www.wikidata.org/entity/Q41892669

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External and internal triggers of cell death in yeast A novel approach to the discovery of anti-tumor pharmaceuticals: searching for activators of liponecrosis Communications between Mitochondria, the Nucleus, Vacuoles, Peroxisomes, the Endoplasmic Reticulum, the Plasma Membrane, Lipid Droplets, and the Cytosol during Yeast Chronological Aging Biosynthesis of oxidized lipid mediators via lipoprotein-associated phospholipase A2 hydrolysis of extracellular cardiolipin induces endothelial toxicity Discovery of plant extracts that greatly delay yeast chronological aging and have different effects on longevity-defining cellular processes Mechanisms Underlying the Essential Role of Mitochondrial Membrane Lipids in Yeast Chronological Aging Empirical verification of evolutionary theories of aging.Mechanisms by which different functional states of mitochondria define yeast longevity.Caloric restriction extends yeast chronological lifespan via a mechanism linking cellular aging to cell cycle regulation, maintenance of a quiescent state, entry into a non-quiescent state and survival in the non-quiescent state.Guidelines and recommendations on yeast cell death nomenclature.Diacylglycerol triggers Rim101 pathway-dependent necrosis in yeast: a model for lipotoxicity.pH homeostasis links the nutrient sensing PKA/TORC1/Sch9 ménage-à-trois to stress tolerance and longevity.Some Metabolites Act as Second Messengers in Yeast Chronological Aging.Yeast Cells Exposed to Exogenous Palmitoleic Acid Either Adapt to Stress and Survive or Commit to Regulated Liponecrosis and Die.Caloric restriction delays yeast chronological aging by remodeling carbohydrate and lipid metabolism, altering peroxisomal and mitochondrial functionalities, and postponing the onsets of apoptotic and liponecrotic modes of regulated cell death.Lipotoxicty in yeast: a focus on plasma membrane signalling and membrane contact sites.Regulated Cell Death as a Therapeutic Target for Novel Antifungal Peptides and Biologics.Mechanisms through which lithocholic acid delays yeast chronological aging under caloric restriction conditions

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Mechanism of liponecrosis, a distinct mode of programmed cell death.

http://www.wikidata.org/entity/Q41892669

description

2014 nî lūn-bûn

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Mechanism of liponecrosis, a distinct mode of programmed cell death.

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Mechanism of liponecrosis, a distinct mode of programmed cell death.

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Mechanism of liponecrosis, a distinct mode of programmed cell death.

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Adam Beach

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Alejandra Gomez-Perez

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Amanda Piano

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Andrew Victor

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Anna Leonov

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Anthony Arlia-Ciommo

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Behnaz Sadri

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Cory Campbell

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Daniel Goncharov

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Dimitri Patrinos

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P2860

Chemical genetic screen identifies lithocholic acid as an anti-aging compound that extends yeast chronological life span in a TOR-independent manner, by modulating housekeeping longevity assurance processes

Classification of cell death: recommendations of the Nomenclature Committee on Cell Death 2009

Autophagic processes in yeast: mechanism, machinery and regulation

Metabolism and regulation of glycerolipids in the yeast Saccharomyces cerevisiae

Spermidine biosynthesis in Saccharomyces cerevisae: polyamine requirement of a null mutant of the SPE3 gene (spermidine synthase).

Role of mitochondria in the pheromone- and amiodarone-induced programmed death of yeast.

The transcription factor Rim101p governs ion tolerance and cell differentiation by direct repression of the regulatory genes NRG1 and SMP1 in Saccharomyces cerevisiae.

The Rim101 pathway is involved in Rsb1 expression induced by altered lipid asymmetry

A novel membrane protein, Ros3p, is required for phospholipid translocation across the plasma membrane in Saccharomyces cerevisiae.

The inhibitor-of-apoptosis protein Bir1p protects against apoptosis in S. cerevisiae and is a substrate for the yeast homologue of Omi/HtrA2.

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