Human cytomegalovirus induces the endoplasmic reticulum chaperone BiP through increased transcription and activation of translation by using the BiP internal ribosome entry site.
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Human Cytomegalovirus Strategies to Maintain and Promote mRNA TranslationCap-Independent Translation in Hematological MalignanciesModulation of the Translational Landscape During Herpesvirus InfectionDifferential unfolded protein response during Chikungunya and Sindbis virus infection: CHIKV nsP4 suppresses eIF2α phosphorylationHCV causes chronic endoplasmic reticulum stress leading to adaptation and interference with the unfolded protein responseQuantitative temporal viromics: an approach to investigate host-pathogen interactionCytomegalovirus downregulates IRE1 to repress the unfolded protein response.Bortezomib induction of C/EBPβ mediates Epstein-Barr virus lytic activation in Burkitt lymphoma.Viral effects on metabolism: changes in glucose and glutamine utilization during human cytomegalovirus infection.The human cytomegalovirus protein pUL38 suppresses endoplasmic reticulum stress-mediated cell death independently of its ability to induce mTORC1 activation.Retroviral vectors elevate coexpressed protein levels in trans through cap-dependent translation.HIV infection and antiretroviral therapy lead to unfolded protein response activation.Analysis of the interaction with the hepatitis C virus mRNA reveals an alternative mode of RNA recognition by the human La proteinMurine cytomegalovirus targets transcription factor ATF4 to exploit the unfolded-protein responseInduction of cap-independent BiP (hsp-3) and Bcl-2 (ced-9) translation in response to eIF4G (IFG-1) depletion in C. elegansActivation status of integrated stress response pathways in neurones and astrocytes of HIV-associated neurocognitive disorders (HAND) cortex.GRP78 at the Centre of the Stage in Cancer and Neuroprotection.Enterovirus 71 induces dsRNA/PKR-dependent cytoplasmic redistribution of GRP78/BiP to promote viral replication.IRES-Dependent Translational Control during Virus-Induced Endoplasmic Reticulum Stress and ApoptosisThe changing role of mTOR kinase in the maintenance of protein synthesis during human cytomegalovirus infection.Functional 5' UTR mRNA structures in eukaryotic translation regulation and how to find them.The Human Cytomegalovirus Endoplasmic Reticulum-Resident Glycoprotein UL148 Activates the Unfolded Protein ResponseBeyond Horses to Zebras: Sicca Syndrome
P2860
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P2860
Human cytomegalovirus induces the endoplasmic reticulum chaperone BiP through increased transcription and activation of translation by using the BiP internal ribosome entry site.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Human cytomegalovirus induces ...... internal ribosome entry site.
@en
type
label
Human cytomegalovirus induces ...... internal ribosome entry site.
@en
prefLabel
Human cytomegalovirus induces ...... internal ribosome entry site.
@en
P2093
P2860
P356
P1433
P1476
Human cytomegalovirus induces ...... internal ribosome entry site.
@en
P2093
Francis J Pierciey
James C Alwine
Nicholas J Buchkovich
Yongjun Yu
P2860
P304
11479-11486
P356
10.1128/JVI.01330-10
P407
P577
2010-08-25T00:00:00Z